Abdominal Pain. Mike Clancy. SEMEP

More goodness from the Southampton Emergency Medicine Education Project Mike Clancy on abdominal pain

Video:

 

Audio:

Or you can download audio here (right-click) or Emergency Medicine Tutorials

A few points of difference between this lecture and Australasian practice:

  • O2 not needed for abdo pain if sats normal.
  • ECG not needed in kids
  • LFT and amylase or lipase on everyone with upper abdo pain
  • CT often before pt s/b surgeon
  • Ischaemic gut: normal lactate does not r/o ischaemic gut
  • I disagree that every abdo pain needs a blood gas.  I don’t do  a blood gas on a young person with mild to moderate abdominal pain.  If you are doing blood gases make sure they are venous and not a cruel ABG.  We can r/o DKA or Addisonian crisis from the electrolytes and a blood sugar.  All extra tests cost money.  Blood gas is not part of the ACEM lab test guidelines, there is just “consider lactate” for severe abdominal pain only

 

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Shortness of Breath. Ian Beardsell. SEMEP

From the wonderful people at the Southampton Emergency Medicine Education Project

Video:

Audio:

or download audio here (right click) or Emergency Medicine Tutorials

A few things we do differently Down Under are that we seldom do ABGs on COPDs, and almost never on asthmatics. A VBG can tell you whether a CO2 is high, low or about normal – and if it is changing over time without repeated painful arterial stabs. More importantly we figure we can tell if a patient is tiring or failing to respond to treatment from the bedside.  Blood gases are more useful for talking to consultants at home who can’t see the patient.

We tend to use more spacers than nebulisers to reduce the spread of infection via the droplets and so patients can learn that spacers work quite well and therefore they can treat themselves at home a lot of the time.

A final point is that a lot of acute mitral regurgitation will settle with treatment of LVF without the need for surgery – but get a big person to have a look at these patients and make the big decisions.

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Syncope. Beardsell. SEMEP

More goodness from the Southamptom Emergency Medicine Education Project

Video:

Audio:

or download (right-click) here or Emergency Medicine Tutorials

Key points (plus some extras from me)

History:

  • Previous episodes
  • Family history of sudden death
  • Hx of cardiac disease – more likely to have a dysrhythmia
  • Syncope during exercise
  • Witnessed convulsion (but may occur with syncope)
  • Standing vs lying (lying much more suspicious)
  • Food intake
  • Preceding symptoms such as feeling faint, hot, nauseated probably more likely benign
  • Preceding chest pain or palpitation more likely to be cardiac or PE
  • Preceding drug use – commonly GTN

Examination:

  • Signs of seizure (not syncope): tongue biting, incontinence, drowsiness/confusion, neurological deficit
  • Signs of TIA/CVA (not syncope): neurological deficit
  • Heart murmur – especially aortic stenosis
  • Brief abdo exam to rule out peritonism, including ultrasound for ?AAA if > 50 years old
  • Signs of CCF – higher risk of cardiac arrhythmia
  • Signs of DVT to suggest PE

Investigations:

  • ECG
  • Blood glucose
  • BHCG in all females of child-bearing age (unless Hx hysterectomy)
  • Hb if pale

Key ECGs

WPW

HOCM

 

Brugada

  • Prolonged QT

Prolonged QT

 

  • One not mentioned in the video: ECG suggestive of PE eg tachycardia and inferior and anterior T wave inversion

PE

  •  LVH on ECG with an aortic stenosis murmur suggests critical aortic stenosis as the cause of syncope.  High risk of sudden death.  Needs inpatient cardiology assessment with view to valve replacement

lvh

Disposition:

In our hospital for a single syncope with low risk history, normal examination and investigations as above, the patient does not get admitted or referred to outpatients clinic – the yield would be too low.

 

 

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Oligoanalgesia

An 8-year-old came into ED with a fractured femur from a school rugby practice. He was about to be sent to the ward with a Sagar Splint on his leg with no traction on it, no femoral nerve block and no opioid charted.

One of our nurses correctly pointed out that we are happy to give you a femur fracture if that is what is required for you to develop empathy with you patients.

If you don’t know how to do any of these procedures, or are too busy, an ED senior will be happy to help out.

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Why I don’t like Digoxin

A 78-year-old woman came to ED with nausea, vomiting and a headache. It seemed to have started during her recent admission to hospital with paroxysmal atrial fibrillation. To be fair to the physicians, they had tried her on decent doses of metoprolol and diltiazem and hadn’t been able to get her rate under control. They had started her on digoxin which finally slowed her down. In retrospect the dose of 125µg daily with a eGFR of 30 was probably a little generous. Always think twice before discharging a patient with a new prescription for digoxin and ondansetron!

On return to ED, her renal function was unchanged and her potassium was her digoxin level came back as 2.96 nmol/L (therapeutic range 0.6-2.0). Many references, including UpToDate and Toxinz, don’t recommend treating chronic digoxin toxicity unless the digoxin level is > 10nmol/L, they are hyperkalaemic, haemodynamically unstable or have life threatening dysrrythmias. Others such as the Toxicology Handbook (co-authored by LITFL’s Mike Cadogan) recommend treating anyone with an elevated digoxin level, renal impairment and symptoms of toxicity eg nausea and vomiting.

This woman was had been treated with several antiemetics and analgesics with little effect. She was lying in bed, looking miserable with intermittent vomiting. 2 hours after being given digoxin immune FAB (eg Digifab) was feeling much better and was sitting up in bed looking as bright as a button.

By the way here is her ECG.

Digoxin

Digoxin

The lateral ST depression suggests digoxin use – but not necessarily toxicity. The classical ECG findings in digoxin toxicity are heart blocks and automaticity, and bidirectional tachycardia. You can also get almost any arrythmia from digoxin. See http://lifeinthefastlane.com/ecg-library/basics/digoxin-toxicity/ for lots of good digoxin toxicity ECGs

I’m currently trying to track down a great talk from Billy Mallon several years ago in which he said “digoxin is a dinosaur looking for a tar pit” which summarised a lot of the reasons ED docs don’t like digoxin.

Another trick to digoxin is not to do another digoxin level when treating digoxin toxicity. The assay measures the bound digoxin and the antibodies binding it and the measured digoxin level doubles – causing much confusion.

Emergency Medicine Tutorials

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Why Amal Mattu doesn’t like amiodarone.

amal-mattu

Amal Mattu is Associate Professor; Program Director, Emergency Medicine Residency, University of Maryland School of Medicine, Baltimore, Maryland. He is the go-to-guy on all things ECG for emergency medicine in the USA, and speaks at all the big conferences. He also replies to emails same day! Here is what he has to say about amiodarone.

Video

If your hospital blocks the video, here is the audio:

or download audio here or here Emergency Medicine Tutorials

Here is a link to the AHA 2010 guidelines Amal discussed in the talk http://circ.ahajournals.org/content/122/18_suppl_3/S729.full

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Why I don’t like amiodarone

Audio

There was an emergency call to CCU.

A 70-year-old gentleman was in CCU being treated for renal failure secondary to angiography to investigate the cause of his dilated cardiomyopathy. He had extensive coronary artery disease but no revascularisation was performed. While eating dinner he went into this rhythm – with no symptoms.

VT

He was then given amiodarone.

Then he developed this rhythm:

Slow AF

with a blood pressure of 40 systolic. That’s when we got called.

He was breathing, able to talk but mentating very slowly, he had distended neck veins but his chest was clear.

The CCU nurses had stopped the amiodarone and were drawing up some atropine.

We gave the atropine and put some pacing pads on him. We waiting a bit because external pacing isn’t pleasant for the patient. We drew up some midazolam and fentanyl in case we needed to pace him.

After 2 minutes his HR was 70 and his BP 85/ with a MAP of 70 and he was looking a bit more awake. Great. (? due to atropine ? due to the amiodarone infusion being stopped)

5 minutes later his BP was 60 systolic with a HR of 72. Pacing probably isn’t going to help much (thanks Bridget). I don’t want him to give him an inotrope as that may put him back into VT. Hmmm fluid load or phenylephrine? We went with 250ml of crystaloid which seemed to do the trick.

Bottom line amiodarone is nasty. This guy should have been electrically cardioverted or given procainamide.

I will occasionally use amiodarone – usually for a really stubborn AF that isn’t converting despite electricity. After 2 shocks I’ll give a 150mg iv amiodarone push then zap again. My record so far is 6 shocks before converting.

But don’t listen to me … next we have Amal Mattu, American emergency medicine ECG guru on the subject of amiodarone.

amal-mattu

Image from http://blog.ercast.org/tag/early-repolarization/

You can download the audio here or here Emergency Medicine Tutorials

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Headache. Sarah Robinson. Southampton Emergency Medicine Education Project

More goodness from the Southampton Emergency Medicine Education Project

The video is here

If you want to download to listen on you bike the audio-only is here (R click to save) or Emergency Medicine Tutorials

To play the audio on your computer if your hospital’s firewall won’t let you watch the video use this:

A couple of things we do differently:

1) we use a CRP rather than ESR to look for temporal arteritis because it’s cheaper and faster
2) we will usually not do an LP if we have a negative CT within 6 hours of onset of a rule out subarachnoid (reference http://emergency-medicine.jwatch.org/cgi/content/full/2011/805/1) but this is controversial.

More on subarachnoid in Life in The Fast Lane here

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The emergency medicine approach to the sick patient

Ian Beardsell (@semep) from the Southampton Emergency Medicine Education Project has kindly given me permission to rip some of the audio from their excellent video introduction to emergency medicine and to share it on this blog and podcast.

I’ll start with Ian’s talk on the emergency medicine approach to the sick or injured patient – how we do things differently in ED.

Here is the original video

Audio-only is available for download either via the iTunes button below or by right clicking here so that you can listen on your bike etc

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Suddenly Swollen

Play audio:

 

 

A 62 year old woman presented to ED within minutes of sudden swelling of her left leg.  Her leg was uncomfortable rather than painful.

Her entire left lower limb is swollen but not firm and it was cyanosed.  Pedal pulses were strong and the leg was warm.

She fractured her left ankle 6 weeks earlier and this was treated with a “moon boot” for 4 weeks.  She has no other medical history, is on no medications and is not a smoker.

She had had no chest pain or shortness of breath.

Obs including blood sugar are normal. Examination was normal other than the swollen blue leg.

Take this quizzes and based on this case and earn some points at the Global Medical Education Project

https://gmep.org/questions/4411

https://gmep.org/questions/4416

https://gmep.org/questions/4415

Scroll down

 

 

 

This is plegmasia cerulea dolens (or dolence) – complete venous obstruction of a lower limb secondary to a DVT in the iliac veins +/- more distal veins.

This is rare (I’ve never seen one, my boss has only seen one) but worth knowing about.  It is not a give-enoxaparin-and-send-home case.  These patients are at high risk of PE, compartment syndrome and gangrene.  Shock may occur from 3rd spacing into the leg.   Overall the death rate is 20-40% with a 12-50% amputation rate among survivors.

This patient did not receive any imaging.  A clinical diagnosis was made, a vascular surgeon was called, IV heparin bolus and infusion started and she was flown to a tertiary hospital for IVC filter insertion and thrombectomy.

If there were no risk factors of DVT it may have been worth taking bloods to screen for thrombophilia prior to starting heparin.  Different haemotologists appear to have different opinions.

Reference: http://www.journal-imab-bg.org/statii-09/content_1/vol-15_book-1_89-91.htm

 
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Insomnia and Sleep

Insomnia appears to be a plague in our society. Many of our patients suffer from it. Many health professionals have trouble sleeping and often use alcohol or other drugs to help them sleep.

Any of the mindfulness meditations in the previous post can be used to help get you or your patients get to sleep – and if it doesn’t work at least you are doing something useful while you are awake. Part of the trick is to not fight the insomnia – or the distractions – while you meditate. Just accept them and turn your attention back to your chosen focus.

Falling Asleep by The Meditation Podcast is a guided meditation specifically designed to help you fall asleep. I gave it to one of my colleagues who was having trouble sleeping. It worked well – but scared the hell out of his wife who got home at 7pm one evening to find her husband asleep on the lounge floor.

Falling Asleep from The Meditation Podcast

Download audio here

Insomnia image from http://www.vhealthjuice.com/wp-content/uploads/2011/06/insomnia.jpg

Garfield sleeping image from http://www.principalspage.com/theblog/archives/sleep-is-a-mysterious-and-complicated-mistress

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Mindfulness for Health Professionals

If you were lucky enough to be at SMACC2013, or have caught some of the talks that have been podcasted or vidoecasted, you hopefully will have heard two of the key-note speakers, Cliff Reid from Sydney and Scott Weingart from New York, talking about the importance of being Mindful in the resuscitation room and meditating daily to help you be a better critical care health professional.

They are talking about old techniques taken from the Eastern meditation practices and being applied to many areas of western life. They are being taught in Masters of Business Administration course, they are being used by sports and business coaches, and increasingly in medicine. These are techniques to help us focus our attention where we want it to be. So in a resuscitation instead of a million thoughts running through our heads “shite, I don’t know what’s going on, this kid is going to die, what will the boss think, what about the patient in the next bay, that surgical reg is an a hole …” etc we can just focus on what our patient needs us to focus on.

Most of us would benefit from spending say 10 – 20 minutes a day meditating by ourselves or with a group to get the hang of it, then slowly and steadily applying these techniques to our work life. We become calmer, more focused and work becomes easier.

To start with, meditation exercises get us to focus on one thing – say our breathing – and we try to keep our attention on that one thing. It comes as a shock to most people how difficult that is – we are lost in thought very quickly. The exercises seem to have little practical application to our work – but with time and practice we are able to focus on our patient in front us, think clearly and not be distracted by the mental chatter.

For most of us it is easiest to use a guided meditation: someone talking to us and reminding us what we are meant to be focusing on. So what follows is a series of guided exercises by Professor Jon Kabat-Zinn, a professor of internal medicine from the USA. He has made a career out of applying these techniques to patients with a multitude of illnesses, and now to teaching business leaders. The exercises are from an audiobook called Mindfulness for Beginners

Try doing one of them once a day for a week, become the next Weingart or Reid!


Breath Meditation by Jon Kabat-Zinn

Download audio here

Image from http://blog.hqh.com/2011/05/13/visualisation-meditation-can-it-help-your-fitness-performance/

 

 

Body Breathing Meditation by Prof Jon Kabat-Zinn

Download audio here

Image from http://neurowissenschaft.wordpress.com/2012/05/13/mindfulness-meditation-improves-cognition-evidence-of-brief-mental-training-2/

 

 

Sound and Thought Meditation by Prof Kabat-Zinn

Download audio here

Image from http://foodmatters.tv/articles-1/7-health-benefits-of-meditation

 

 

Nothing Meditation by Prof Kabat-Zinn

Download audio here

Image from http://www.weekendnotes.com.au/im/005/07/meditation-courses1.jpg

 

 

Emergency Medicine Tutorials

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Major Burns

I’m just back from an Emergency Management of Severe Burns course.

Great little course. Concise manual. A day of very brief lectures, then skills stations, then scenarios then a written and practical exam. It runs in 16 countries including Australia, NZ, UK and South Africa.

Not everyone will be able to go on the course tomorrow so I’m going to cram the key points into 20 minutes for you.

A big focus of the course is to ensure the ABCs are assessed and reassessed, and a reminder that a lot of severe burns are potentially multitrauma.

They have adapted the standard trauma primary and secondary survey by putting First Aid between the primary and secondary survey. First aid includes 20 minutes of cooling which can be effective up to 3 hours post burn (which was new to me). And they used the mnemonic FATT for the first aid component: fluid, analgesia, tests and tubes. I’ll go into this in more detail soon.

An approach to the Severely Burned Patient

An approach to the Severely Burned Patient

For a moderate/severe burn:

A: includes C-spine control if there is a chance of trauma. The airway assessment particularly looks for signs of potential airway burn: singed nasal hairs, soot in the mouth, change in voice, stridor, sooty sputum. If there are any of these signs get a senior to consider urgent intubation.

B: Looks for signs of trauma eg pneumothorax, signs of inhalational injury eg increased work of breathing, hypoxia, creps. If there is any chance of significant exposure to cyanide or carbon monoxide ->high flow oxygen – and this is one situation where we don’t wont to taper the oxygen quickly. Oxygen sats may be normal in significant carbon monoxide poisoning. Inhalation injury (lung damage) may develop quickly – keep reassessing the patient’s lungs. If there decreased chest movement because of tight burned skin an escharotomy will be required.

Escharotomy Lines

Escharotomy Lines

C: HR, BP, capillary refill (they were keen for an unburned digit – i think a central CRT is generally accepted as a better measure of circulatory status). Look for other sources of shock if signs of shock eg external haemorrhage, EFAST. Big IV lines (through unburned skin if possible). Use an IO if necessary. Bloods including carboxyhaemoglobin, lactate, and G+H, FBC, U+E, BHCG, BSL.

D: AVPU and pupils

E: Remove all jewelry and piercings as they may be hot and keep burning, or may cause problems as the patient swells. Remove all clothing. Log roll now to check the back and see the extent of burns. Cover the patient and prevent hypothermia.

First aid:

Cooling: Cooling for 20 minutes, if not already done, can be effective up to 3 hours post burn. For a big burn you might be able to put the patient in your decontamination shower and cool them with water at about about 37˚- cool enough to cool wounds, but hopefully won’t cause hypothermia

Fluids: 3-4ml x kg x % BSA burned (excluding epidermal burns (erythema without blistering ie looks like unblistered sunburn) over 24 hours with the first half within 8 hours of the time of burn (not ED arrival). They are keen on Hartmann’s/lactated Ringer’s for this resuscitation fluid. With kids give standard maintenance fluid (eg 1/2 normal saline with 5% dextrose) at standard rates through a separate cannula (could be piggy backed if necessary). More discussion re fluids later.

Analgesia

Tests – blood tests if not already taken, and what ever trauma imaging is required if not already done

Tubes – NG tube if the patient is being transferred by air or if over 10% BSA in kids or 20% BSA in adults – to decompress the stomach early, and then to start NG feeding within 6 hours.

Then their description of a secondary survey is:

AMPLE history (I always stick a T at the end of this to remind me about tetanus, I know AMPLET doesn’t roll off the tongue but some times the sillier the mnemonic the better I remember it). In the Events we particularly want to know the details of the burn: when, duration of the burn, type of burn (eg what chemicals), clothing worn / burned, temperature of the fluid (eg water at max 100˚C or canola oil at max 800˚C), first aid before ED.

Head to toe examination looking for extent of burns and complications of burns eg decreased peripheral circulation or sensation due to eschar (burned skin) or compartment syndrome. Also looking for associated trauma.

Documentation: eg use a referral form from your local burn unit eg http://www.nationalburnservice.co.nz/pdf/referralform.pdf

Reevaluate

Arrange transfer to a burns unit for definitive therapy.

Glad wrap / plastic film is a great dressing for patients being transferred.

Explanation and psychological support for patient and family.

So that’s the general approach.

Now some more detail.

Fluid resuscitation: I took the opportunity to ask the burns gurus where we are out with fluid resuscitation for burns. I know after the Bali Bombings and the Ashmore Reef disaster the Darwin legends were keen on less generous fluid resuscitation as they believed they got into less problems with airway swelling and other problems with oedema. The current thinking is that the fluid replacement needs to be reassessed, individualised and fine tuned. The formulae just give a starting point. If the urine output is significantly greater than the targets of 1ml/kg in a child or 0.5ml/kg in an adult, the fluids should be slowed thus reducing the risk of oedema. Reassess hourly.

Burn depth estimation. Burn depth estimation is inaccurate and apparent burn depth can change over the first few days. For initial resuscitation we only really care about burns deeper than epidermal, but we need to be able to describe burns to our colleagues.

We need to describe colour, blistering, capillary refill, pain, exudate, surrounding inflammation, size and distribution.

In a nutshell there are epidermal burns (erythema only), dermal and full thickness burns. Dermal burns are broken down into superficial, mid and deep dermal. Superficial dermal is usually associated with blisters (but blisters may have burst and the epidermis may have gone) the base will be uniformly red, and will blanch, it will be painful and have sensation. Mid dermal will be similar but with darker pink base, may or may not have sensation and will have sluggish capillary refill. Deep dermal burns are blotchy red, with no capillary refill and no sensation. Full thickness burns are charred or white with no capillary refill and no sensation.

As burn depth can change over the first few days we shouldn’t attempt to prognosticate.

Beware of circumferential burns – may need a escharotomy.

Elevate any limb with significant burns especially if impaired circulation or marked swelling.

If the patient is oedematous in both arms get an arterial line in early to assess BP.

Haemo or myoglobinuria (usually from high voltage electricity) requires increased IV fluids aiming for urine output of 2ml/kg/hour.

Burn Centre referral criteria

· Burns greater than 10% total body surface area (TBSA) or 5% in kids

· Burns of special areas, e.g. the face, hands, feet, genitalia, perineum, and major joints.

· Full thickness burns greater than 5% TBSA.

· Electrical burns (including lightning injury).

· Chemical burns.

· Burn injury with inhalation injury.

· Circumferential burns of the limbs or chest.

· Burns at the extremes of age, i.e. young children and the elderly.

· Burn injury in patients with pre-existing medical disorders that could complicate management, prolong recovery, or affect mortality.

All electrical burns should have an ECG on arrival. High voltage should have 24 hours of telemetry.

Patients with electrical injuries can recover well after prolonged CPR – so don’t stop!

There is a new chelating agent called diphoterine which can bind many toxins including acids and alkalis. Very good for eyes. I’m tracking some down for our ED.

Some specific chemicals we need to know about:

Hydrofluoric acid: a 2% BSA exposure can kill. Good antidotes.

Cement: Can cause severe burns hours after exposure -> prolonged irrigation.

Bitumen: Irrigate till well cooled but don’t panic, it’s not toxic.

Petrol: Can cause renal and hepatic toxicty

Alkalis: Required longed irrigation

Minor burns

Blisters. No firm opinions but generally leave small ones alone. Deroof larger ones – partly to allow assessment of the underlying burn depth, partly to avoid the patient panicking when the blister bursts.

Wash all burns.

Use silver impregnated dressing if burn was contaminated. eg acticoat (TM) soak in water, apply silver/blue side to skin, put a damp gauze over top, then adhesive dressing over top. Otherwise use silicone dressings eg mepilex, hydrocolloid eg duoderm, films eg opsite or tegaerm.

SSD (Silver sulphadiazine) is out of fashion for moderate/severe burns as it needs to be applied twice a day to keep silver levels therapeutic and it discolours tissues making burn depth assessment difficult.

Follow up burns at 2-3 days.

Surrounding inflammation suggests infection – discuss with burn unit as infection can worsen burn severity

Frank Burns image from http://welcometothe4077.tumblr.com/

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Aortic Emergencies for Junior Docs

Here’s a talk from Professor Wayne Triner from the United States who we are lucky enough to have working in our little ED.

Hopefully you will have already listened to Scott Weingart’s talk on EMCRIT on Mx of Aortic dissection. There are a few differences in opinion on which drugs to use, and Scott talks about some drugs that I’m pretty sure we can’t get in New Zealand. My choices of initial drugs would be fentanyl, labetalol and IV diltiazem just because I am comfortable using these drugs. Later I can talk to cardiology and cardiothoracics and see what they want used.

Now for Professor Triner

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Airway Foreign Body

A toddler is brought into ED after a choking episode while playing on the floor at home.

He is now undistressed and there is no abnormality on examination. Click on the image to enlarge it.

Choking episode

 

How will you manage this patient?

Take the quiz at the Global Medical Education Project: https://gmep.org/questions/4359
(Sign in in the blue button on the right of the screen: Sign in to see if you have the correct answer. It is well worth joining the Global Medical Education Project: a fabulous, fantastic, free, learning resource)

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NZ Medical Law and Ethics Update 2013

Highlights from 14th Annual Medical Law Conference, Wellington, March 2013

Health and Disability Commissioner case of an inexperienced GP who prescribed propranolol for migraine to a patient with moderate to severe asthma. Patient died. So a reminder to check patients’ medical history

http://www.hdc.org.nz/decisions–case-notes/commissioner%27s-decisions/2006/04hdc19938

Presented by Dr Chris Cameron, Clinical Pharmacologist and General Physician, Wellington Hospital

 

 

To break confidentiality now need a serious threat to patient or others, rather than serious and imminent.

Presented by Sebastian Morgan-Lynch, Senior Policy Advisor (Health), Office of the Privacy Commissioner.

 

Centralised coroners service available 24/7. If you have any question whether a case should be referred to the coroner phone them 0800 266 800. Must notify coroner if medical error could have contributed to the death.

Presented by Judge Neil MacLean, Chief Coroner

 

Need to report medical errors – your own or other health practitioners via accepted reporting mechanisms. Eg hospital’s incident reporting system. You could be found guilty of misconduct if you don’t. But also acknowledged that risk management systems can be over whelmed by reporting of minor incidents. If in doubt discuss with a senior colleague.

Presented by Helen Brown, Lawyer, Kensington Swan.

 

2012 Amendment of the Crimes Act means potentially tougher penalties for failure to report abuse or neglect of a vulnerable person. Unlikely to be used against a health professional.

Presented by Iris Reuvecamp, Lawyer, Claro Law.

 

Common cases before Health Practitioners Disciplinary Tribunal: Sex with patient or possessing pornography.

Presented by David Carden, Barrister, deputy chair HPDT

But penalties seem weak eg 18 months suspension + rehabilitation, for storing 400,000 images of child sexual abuse.

 

Further debate re allowing suicide of a terminally ill patient or one with a severe degenerative illness. At last years conference there was discussion of a hypothetical case of 50-year-old male with terminal cancer who was brought in by ambulance with a suicide note saying he had taken a drug overdose. Professor (of medical Law) Skegg had suggested it would be acceptable to let this person die. It subsequent discussion in my institutions many senior doctors who horrified by this. So this year Prof Skegg suggests that the standard of care is the what a group of doctors would do – and so, given the reaction of the doctors in my workplace, active treatment would be required. An ED physician from Middlemore suggested that the appropriate treatment would only be the equivalent of what would be given to palliative patient who hadn’t suicide die we would be unlikely to intubate and ventilate the patient – but we might give them other medications eg antidotes to the overdose agent. If the situation came up it would be a high level decision – a couple of senior doctors, possibly including a psychiatrist to help with the decision-making. It would probably be wise to discuss with your medical indemnity insurer as well.

Discussion with Professor Skegg, Professor of Law, University of Otago

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SMACC 2013

Social Media and Critical Care conference 2013 March 11-13 in Sydney.

This will be the international cutting edge of ED, ICU, anaesthesia, rural and pre-hospital education with Scott Weingart from EMCRIT (currently having 100,000 downloads a month!) , Mike Cadogan and Chris Nickson from Life in the Fast Lane, Cliff Read from Resus.ME.  The American Academy of Emergency Medicine has named their Educator of the Year Award the Joe Lex Award after Joe Lex, he’s a legend of EM teaching and you will have heard him on EMRAP, also runs Free Emergency Medicine Talks – he’s coming.  Simon Carley who helped set up BestBets and the StEmlyns Blog will be there as will be all sorts of other clever people.

Most of us antipodeans don’t know how lucky we are to have this conference – and so much of the cutting edge of emergency / critical care education – coming out of Australia.

These guys will be presenting the latest in ED and ICU knowledge and it will be crazy fun!  We’ll be laughing as we are learning.

Bored with conventional medical education?  This is for you!

There are all sort of innovative educational tidbits including SIMWARS, Sono-games (test you ultrasound skills), and PK SMACC (Pecha Kucha) – put together a 6 minute talk – send it in and you might get to present it at the conference.

Early bird registrations close 1 February 2013

 

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Down the K Hole via the A Hole

 

Following on from Cliff Reid’s talk “Scoop and Run is for A Holes “yesterday at Essentials of Emergency Medicine, today we Michael Levine  gave a talk that could have been called “Down the K hole via the A hole”

His talk was on alternative routes of administration for sedative and anaesthetic drugs.

Personally, when I haven’t got an IV line, I like fentanyl IN but otherwise I like to drugs IM so I know how much has actually been absorbed.  Unless it’s a really short procedure I’ll usually place an IV line once the kid is under because if you have to redose IM the patient will sleep for hours.

DRUG

INTRANASAL DOSE

Fentanyl

1.5-3 mcg/kg

Ketamine

8-9 mg/kg

Midazolam

0.5 mg/kg

Naloxone

0.005-0.1mg/kg

Sufentanil

0.2-1 mcg/kg

 

It’s good to see that fentanyl dose is creeping up.  A few years ago it was 1.5µg/kg which never seemed to cut it, then it was 2µg/kg which was sort of OK, but now 3µg/kg is sounding better).  And the few times I’ve used IN ketamine it’s been pretty rubbish – but I was using 5mg/kg so I was under dosing.

(Note: Michael Levine left the nalaxone dose blank.  I’ve added the dose from Pedistat.  The lower dose is for reversal of anaesthesia, the highest dose to reverse an overdose)

But I hadn’t even thought about giving rectal ketamine.

 

DRUG

RECTAL DOSE

Diazepam

0.75 mg/kg

Ketamine

10 mg/kg

Methohexital

25-30 mg/kg

Midazolam

0.3-1 mg/k

 

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Hip Relocation

Inspired by a talk by Billy Mellon at Essentials of Emergency Medicine 2012 today

Captain Morgan technique

Whistler Technique BUT Billy says the proceduralist turns her/his back to the patient, bends her/his knees, then straightens her/his legs to lift the patients knee.  DO NOT put force down on the tibia because there have been cases of tibial fracture from this!

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He Can’t Breath on the Ventilator – Cliff Reid

The third and last talk by Cliff Reid of Resus.me this time on managing the ventilated patient that you can’t ventilate.

Disconnect from ventilator, let them exhale, in case there is breath stacking

ETCO2.

Hand ventilate.

Reintubate if flat trace.

Difficult to bag:

  • Suction the tube
  • ? tube too deep and causing bronchospasm -> withdraw tube a few cm
  • ? tube down R main bronchus ->
  • Pneumothorax: bed side ventilator

Too easy to bag = leak eg cuff leak, tube not through cords.  -> reinsert or replace ETT over bougie

Fighting the vent -> more analgesia and sedation +/- paralysis

Lung disease -> keep plateau pressure < 30cmH2O.  TV usually 6ml/kg IBW.  May need to increase PEEP to recruit alveoli.  Avoid derecruitment when changing circuits / ventilators: clamp the ETT.

Asthma and COPD: small tidal volumes, long expiratory time, low rate.  Permissive hypercapnia.  Low or zero PEEP.  If decompensates disconnect and let them exhale.

Extrinsic compression eg

  • Obesity -> ramp,
  • Full stomach -> gastric tube
  • Abdominal compartment syndrome -> surgeon.  May be helped (temporarily while waiting for surgical decompression) by increased analgesia +/- paralysis
  • Gravid uterus -> L lateral position

Back to the circuit: eg leak eg a suction port open, PEEP valve set too high.

 

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Scoop and Run is for A Holes – Cliff Reid

 

Another great talk from Essentials of Emergency Medicine 2012 – today. Cliff Reid from Resus.me again. This time he is debunking some of the myths (and my beliefs) about prehospital stay and play vs load and go.

Stay and play, +/- RSI, has actually been shown to get patients to theatre more quickly. And with less and less trauma being managed operatively, the rush to get patients to theatre is not so great as it used to be.

Our ambos are going to love this one.

:-)

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Own the Resuscitation Room – Cliff Reid

Cliff Reid from Resus.me gave this talk at Essentials of Emergency Medicine in Las Vegas today.

He discusses mastering your team, yourself and the patient.  He advocates training in resus for resus, having systems to manage stress like RSI checklists

Here’s the one we use in Whanganui:

It looks complicated – because it’s a complicated process – and there is a lot to prepare and do right to make sure it goes smoothly. It also provides great documentation of what actually happened when.

Sneaky little screen shot of Cliff’s causes of shock:

[Hmm.  Still gotta get sepsis, anaphylaxis and toxins in there some where.]

We need to encourage our teams to help us / challenge us / remind us of things we may have forgotten or when we are heading down the wrong track.  The team leader should keep their hands off the patient and avoid becoming task focused.

Control the environment.  Don’t allow the environment to control you.

We need to control the mob of helpers.  Get everyone on the same page by regularly verbalising assessments and plans.  We need to ask individuals to do tasks not just float a request out into the room.  Different teams will be focused on their “bits” eg the surgeons on the belly.  We need to keep the over view.  We need to learn graded assertive techniques and to learn the science of human persuasion.  Cliff sagely notes this doesn’t work well at home.  Give annoying people a job eg ask the surgeon to do a cut down (while one of your team puts in the IO in a fraction of the time) ;-)

We need to be comfortable with allowing patients to die with dignity when this is appropriate.

We need to learn from the cases that don’t go well.  Weingart: “A good resuscitationist agonises.”

Ah, a man after my own heart.

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