Own the Resuscitation Room – Cliff Reid

Cliff Reid from Resus.me gave this talk at Essentials of Emergency Medicine in Las Vegas today.

He discusses mastering your team, yourself and the patient.  He advocates training in resus for resus, having systems to manage stress like RSI checklists

Here’s the one we use in Whanganui:

It looks complicated – because it’s a complicated process – and there is a lot to prepare and do right to make sure it goes smoothly. It also provides great documentation of what actually happened when.

Sneaky little screen shot of Cliff’s causes of shock:

[Hmm.  Still gotta get sepsis, anaphylaxis and toxins in there some where.]

We need to encourage our teams to help us / challenge us / remind us of things we may have forgotten or when we are heading down the wrong track.  The team leader should keep their hands off the patient and avoid becoming task focused.

Control the environment.  Don’t allow the environment to control you.

We need to control the mob of helpers.  Get everyone on the same page by regularly verbalising assessments and plans.  We need to ask individuals to do tasks not just float a request out into the room.  Different teams will be focused on their “bits” eg the surgeons on the belly.  We need to keep the over view.  We need to learn graded assertive techniques and to learn the science of human persuasion.  Cliff sagely notes this doesn’t work well at home.  Give annoying people a job eg ask the surgeon to do a cut down (while one of your team puts in the IO in a fraction of the time) ;-)

We need to be comfortable with allowing patients to die with dignity when this is appropriate.

We need to learn from the cases that don’t go well.  Weingart: “A good resuscitationist agonises.”

Ah, a man after my own heart.

Emergency Medicine Tutorials

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ACEM Curriculum Review

I was disappointed to read that the Australasian College for Emergency Medicine’s Curriculum Revision Project looks set to retain the traditional Primary Exam with anatomy, physiology, pharmacology and pathology papers.

I am sure the issue is more complicated than I appreciate but to get a little discussion going I would like to share an open letter to the College.

Hello

We need a green fields approach to this review, especially Primary Exam (aka Part One), rather trying to make anatomy, physiology, pathology and pharmacology look clinically relevant.

The core skills I need my junior trainees to have is: how to read an ECG like an ED doc, how to take a history like an ED doc, how to examine a patient and interpret the findings like an ED doc. Basic airway management, basic breathing management, basic circulatory management, etc. An approach to: chest pain, SOB, decreased LOC, shock, abdominal pain, trauma, anaphylaxis, asthma, COPD, “dizziness”, syncope, stoke, bleeding in early pregnancy, mass casualty, the poisoned patient etc etc.

In a clinically focused training they will learn some relevant anatomy, physiology etc but let’s make the training and the trainees fit for purpose.

Yes it will be harder to examine and to mark.

The proposed CRP looks like it will keep the trainees jumping through hoops, learning stuff to pass exams and continue to distract them from learning real emergency medicine and thus from helping our patients to the best of their abilities.

Emergency medicine should be leading the way in medical training not following along what the other colleges have always done.

Regards

Dr Chris Cresswell

If you have some feelings on this matter I suggest you email Ruth.Hew@acem.org.au with your submission.

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Highlights Essentials 2012 Day 1

 

 

I was up at 4 to catch the start of  Essentials 2012 in Los Vegas @ home. For those who don’t know Essentials of Emergency Medicine is a four-day emergency medicine conference run by Mel Herbert and the good people from EMRAP.

Now as you would expect from the EMRAP team this is succinct, punchy, super relevant 5, 10, 15 minute talks about what we really want to know on the floor at work in an ED, and they make it available online.

It’s pretty cheap for residents / RMOs at $170 USD or $206 NZD, and $850 USD or $1100 NZD for consultants you get to go to an excellent international emergency medicine conference, not have to leave your partner or family, and not have a carbon foot print the size of New Zealand flying there.  And you will be up to date with (almost) everything you need to pass exams and more importantly to be a good doctor for your patients.  For the consultants of course it’s easy CPD points.

Technology is still trying to keep up with these boys and girls who are pushing the international education envelope and we lost the stream several times today but some of the high lights for me:

  1. No fever -> no blood cultures, home.
  2. Fever -> blood cultures, antibiotics, admit
  3. Abdo pain, fever: may be Typhlitis = neutropaenic enterocolitis = surgical emergency -> CT abdo and urgent surgical consult
  • Tumour lysis syndrome: never sticks in my head because I’ve never seen one (or have never recognised one).  Usually seen in the first 5 days of oncology treatment, but can also occur in untreated cancers.  High cell turn over causes hyperuricaemia, hyperphosphataemia and renal failure with hypercalcaemia.  Mx: treat hyperK, fluids +++, cardiac monitoring, correct other electrolyte abnormalities (but treat hyperPO4 before giving Ca2+), needs ICU bed and repeat electrolytes q 4-6 hours
  • ACE inhibitor induced angioedema.  Give 2-3 units of FFP and watch very carefully, if progressive of tongue swelling intubate early.
  • Penetrating foot injuries.  If nail has gone through eg rubber soled shoe go hunting for imbedded rubber.  Tetanus booster if due. No prophylactic antibiotics.  If still sore in a week: MRI and probable operative exploration.
  • Mobile Health: phone based patient education / motivation / reminders.  Phone ownership high across all racial and socioeconomic groups.  eg one program to educate young mums sends out an education snippet three times a week.  As some of you will know I’m a meditation junky … and yes, there is an app for that eg Mindfulness TS
  • Then there was a 2 hours session on the finer points of ECGs that we need to know: how do you tell early repolarisation from STEMI.  Early repol:
  1. In V1-V4,
  2. There will usually be sharp deflection after the J point in V4 (“Fish hook”),
  3. Other than the fish-hook the J point should be a gentle curve into an upwardly concave (smiley face) ST elevation.  If you see this it is early repol or hypothermia.
  4. A sharp, almost right angle J point is more suggestive of STEMI
  5. Early repol will not have reciprocal ST depression in other leads.

 

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Mass Casualty / Coordinated Incident Management

We had a gas leak at a local factory with 2 workers GCS 3 at the scene and 21 others exposed to the gas.

This gave us a chance to test our new Mass Casualty plan which had been developed largely by super ED nurse Michelle Batterbee. Tip for anyone else doing this work get a slightly OCD nurse to drive it not some ADHD ED doctor who won’t get deep enough into the detail.

A few Cordinated Incident Management System buzz words for exams: the 4 Rs: Reduction, Readiness, Response, Recovery

Reduction: what happens out in the real world by to prevent disasters
Readiness: planning and training to deal with a disaster
Response: what we do if a disaster happens
Recovery: getting back to normal, including lessons learned from the disaster.

We received a phone call from ambulance control at 5pm on Friday night “Major incident at the tannery. 2 stat 1 and 13 stat 3. No further information available.”

We rang ambulance control back to check this was correct and if there was any more information. It was and there wasn’t.

Probably going to be a chemical exposure with 2 sickies and a lot of minors with minor exposure.

Now I’m into the 5 Ps

People: I’m keen to call a mass casualty incident. My boss, the wise Athol Steward, says hang on till be get a bit more info – let’s see if they are “real” stat 1s. There are different systems for declaring a mass casualty incident. We use a 3 level system.

Level 1: we should be able to manage in ED with a bit of extra staff and maybe the ability to overflow.

Level 2: this is going to stretch the whole hospital.

Level 3 we are going to need help from other hospitals / military etc.

Which level is declared will depend on the current state of the department, time of day, size of hospital. A level 3 for a small hospital at 2am may be a level 1 for a big hospital, in the middle of a quite day. We are a 15 bed + 5 obs beds ED so 15 patients are going to swamp us.

Inform the team and assign roles. To start with one nurse, one consultant into 2 resuscitation rooms. Other staff doing what they can to clear ED.
We’d spoken to the on call anaesthetist who was just starting an operation. :-(

Personal Protective Equipment

We have a recently obtained a HAZMAT suit – um – anyone know where it is? Who should wear them – just the guys doing the decontamination? We compromised with long-sleeved plastic gowns and some of us put on goggles and masks.

I’ll do a separate talk on the HAZMAT aspects of this case later.

Place: Clear some space: Get patients admitted, discharged or out to the waiting room or discharge lounge now. Clear the resus bays. Put a sign out in ED warning of a long wait. Alert the GP clinic next door that we are about to get slammed. Consider ambulance bypass if that is an option at your hospital.

Clear out all the crap that is being stored in the decontamination shower. Remind yourself how to turn it on :-)

Plant (=equipment): What equipment and drugs to we need? Start setting up airways trolleys and drawing up anaesthetic drugs.

Plan: Start talking through plans, who will do what based on what you know.

First patient arrives, unresponsive, laboured breathing. The ambos just scooped and ran with him. He stinks of sulfur and we hear that sulphuric acid and sodium sulphate were involved. A quick wash in the decontamination shower trying to not to drown him. We hear the next patient is already intubated. The decision was made to call a Level 2 mass casualty incident and to call for 2 anaesthetists.

The beauty of this is that we make one phone call the telephonist who then goes through a list of all the people who need to be called and we can concentrate on what is happening in ED. And you get a lot of extra help. Physicians and surgeons appear in ED and admit or discharge patients. Beds are miraculously found on wards. Extra orderlies / porters appear, extra cleaners, the kitchen starts making sandwiches, the media liaison takes all the medial calls. Extra ED nurses come in (luckily it was early in the evening they were still sober). Extra bed managers come which is essential. Pharmacists turn up with drugs galore. Extra linen appears. We also called in off duty ED consultants (thanks Stuart). And in these situations people are keen to come in to play and to help.

The Mass Casualty box is opened

 

It contains lots of folders with “Unknown patient” labels (if needed), wrist bands, a paper based ED chart, lab forms, XRay forms. Patient tracking forms, signage, simple instruction cards. The patients had a ID label placed on their wrist and forehead with a hand written nickname “One”, “Two” added to it for ease of communication.

Staff wear vests to identify jobs to make it easier for newcomers to work out who is who.

Only ED staff get the vests. Staff from other wards that come to help wear their own uniforms.

If needed we make up teams of 1 ED doc, 1 ED nurse + a ward nurse and a ward doctor. During the big Christchurch earthquake 2 years ago the teams were ED doc, ED nurse, anaesthetist + another nurse + a medical student as a runner. This apparently worked very well. it is really important to have a doctor and nurse who are ED trained and know how ED works in each team. Everyone gets a job card telling them who they report to. So it states that the ward doctor reports to the ED doctor in their team. The ED doctor reports to the ED doctor in charge etc.

In our little department we assign a place for a nurse coordinator, an extra triage nurse, a transport nurse, an orderly.

When extra ED consultants arrived they joined the triage nurses at the ambulance door and at the waiting room triage area and worked with the triage nurses to triage and rapidly assess patients. For the minor chemical exposures this is a quick history, obs and listen to the chest. Many of the minors patients could be held in the waiting room to go directly to an obs ward for their 3-4 hours of observation.

As more senior help arrived our head of department stepped back from direct patient care and became Doctor in Charge of ED He coordinated the medical response and liaising with senior nursing and management.

Our patient assessment and management in a mass casualty situation is pretty much business as usual. We do the usual ED focused history and examination. Maybe a few less tests, less social history but otherwise do everything you would normally do. The main differences are in logistics – not in patient treatment Make sure everything is documented otherwise things turn to custard. Normally you will have enough people coming to help that you wont be short-staffed. Make sure the normal channels for patient tracking and admission are followed or patients will get lost.

The first patient was intubated and ventilated. The second one, who had had a respiratory but not cardiac arrest at the scene, arrived intubated and just had to be washed and tidied up a bit. We eventually found out the gas that had been created by the chemical spill was hydrogen sulfide and administered sodium nitrite and an antidote – causing profound hypotension with a bounding pulse in one of the patients.

And we had 19 minor patients including a whole shift of paramedics and a couple of firies who had been exposed to varying degrees to the gas.

The two sickies were flown out because our ventilators were already full of old crumblies. We had all but 3 of the others discharged within 4 hours – leaving us an almost empty department. It is amazing how the hospital could accommodate all our other patients. Then the recovery. Restocking, cleaning up and debriefing over pizza donated by a member of the public. Then more formal debriefs – how could things be improved – in days to come.

One lesson learned for me was that I didn’t consider the possibility of a blast. We weren’t told there was a blast but I didn’t specifically ask about one. When someone suggested it to me a few minutes later, the paramedics had already gone and I couldn’t get the necessary information from ambulance control. So we treated the 2 sickies as if there could have been a blast and did a panscan CT to be on the safe side.

In retrospect you could say that calling this a mass causality incident was an over call but
a) It’s better to over call than under call and you can always stand down the incident if it turns out it wasn’t as bad as you thought
b) With that volume of patients in our little ED it was good to have the extra staff.
c) It’s good to use the mass cas. plan every now and then so people are familiar with it – and so we can test and fine tune it.

Ours worked well. Thanks Michelle!

Emergency Medicine Tutorials

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When things go wrong

 

 

We are human.  We all stuff up sometimes.

We try to prevent mistakes by studying, peer review, courses and listening to our colleagues – especially our patients’ guardian angels, the nurses.

But when we do make mistakes  what we do afterwards to address the mistake is important.  As I’ve made plenty of mistakes I’m now getting pretty good at this ;-)

So let’s look at a recent case of mine.

An 85-year-old lady with a skin tear of her shin is referred in by the wound care nurses because the wound is growing a group B strep and the wound is smelly. I’m not a great fan of wound swabs, especially in our low MRSA environment, because wound colonisation does not necessarily mean infection.  An RMO / resident assessed the patient and asked me to have a look at the wound.  With my bias against wound swab I went to assess the patient.  Post cleaning the wound looked not too bad, it wasn’t sloughy, it didn’t smell, it had a little redness around it, it didn’t look infected but there was some dead tissue in the wound.  I thought it needed debriding but not antibiotics.  I debrided the wound using IV fentanyl as analgesic. The wound looked quite healthy after the debridement.  I arranged for dressing and follow up next day with no antibitoics.  The wound care nurse then approached me saying the woman was on immunosuppressants for her rheumatoid arthritis and needed antibiotics.  I hadn’t been aware of the immunosuppresants but stubbornly persisted with my plan.

A few days later the woman returned with definite infection of the wound and ended up admitted on IV antibiotics.

In the investigation of diagnostic errors several types of errors have been described:

•          Anchoring bias – locking on to a diagnosis too early and failing to adjust to new information.

•          Availability bias – thinking that a similar recent presentation is happening in the present situation.

•          Confirmation bias – looking for evidence to support a pre-conceived opinion, rather than looking for information to prove oneself wrong.

•          Diagnosis momentum – accepting a previous diagnosis without sufficient skepticism.

•          Overconfidence bias – Over-reliance on one’s own ability, intuition, and judgment.

•          Premature closure – similar to “confirmation bias” but more “jumping to a conclusion”

•          Search-satisfying bias – The “eureka” moment that stops all further thought.

I anchored that this leg was not infected.  We do similar things with our treatment decisions:  I anchored onto my treatment plan

 

When first informed about this turn of events, I had that sick feeling in my stomach we all know, then I got defensive and tried to justify what I had done, but slowly I accepted what I’d done was just wrong.

 

So what do you do when you make mistakes?

As a junior the first thing to do is to talk to a senior

a) to let them know,

b) to get some perspective on what you have done.  Some times we flog ourselves when really our mistake was only a tiny factor in what went wrong, or for the more  arrogant among us we may underestimate the impact of our mistakes, and

c) so they can guide your response to this mistake.  Your response will be guided by your medico-legal environment.  I am lucky enough to work in New Zealand where we have a system that allows and encourages early apologies.

For a more serious case you will be going through your hospital’s complaints/patient safety system and involving your medical defense organisation and will follow their advice.

Many minor cases can be addressed with an apology to the patient.  Go to the ward or phone the patient at home.   The approach for exams and lawyers is to express sympathy without admitting fault: “I am sorry this has happened to you.  We will be investigating what happened and will let you know the outcome.”  Often, however, we know that we were wrong and the best thing to do is to acknowledge this and apologise.  Patients are usually incredibly generous and forgiving.  They can see you are a caring human who is actually remorseful and this does a lot to make them feel better.  You were not some heartless doctor who doesn’t care that she ended up with an infected leg.  If the patient does not forgive you, well at least you know where you stand and you feel a little better for trying to apologise.  Most of the time though, patients do accept our apologies, as this lady did, and this is a huge weight off your shoulders and you can get on with your work without the guilt hanging over you.

Write in the patient’s notes that you have apologised, or expressed your sorrow about the patients situation without acknowledging guilt, or what ever you did:

a) this an important legal record, should this case end up in court

b) it lets the inpatient team know you have made the effort to communicate with the patient and have learned from your mistake.

Also apologise to anyone else that you need to.  In this case I needed to apologise to the wound care nurse.

The case went into our hospital’s incident reporting system for further investigation as required.

Last thing is to share what you have learned with your colleagues.  Hopefully you have a regular departmental meeting where cases are discussed and you are encouraged to talk about cases that went wrong and what you learned from them.  If you don’t have one of these meetings make one happen!  It was one of my great pleasure to arrive at my current department and find they had weekly meetings in which the discussed interesting cases and lessons learned.  An environment that encourages open disclosure to patients and to colleagues is wonderful to work in – and safer for patients.

 

Reference:

Croskery.  2003  www.ncbi.nlm.nih.gov/pubmed/12915363

Emergency Medicine Tutorials

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Video laryngoscopy should be “standard of care”

Hopefully you will have been following the video laryngoscopy standard of care debate on EMRAP. If you haven’t, airway guru Ron Walls stated a few months ago that video laryngoscopy was the standard of care. He got slammed and had to back up a bit.

My much less expert opinion is with Ron. While video laryngoscopy is not standard of care yet, it should be. If you have a video laryngoscope, eg a Glidescope or C-Mac, in your department but chose to use an old-fashioned laryngoscope and there is a failed intubation and a poor outcome how are you going to defend yourself and how are you going to live with yourself?

And if you haven’t got a video laryngoscope in your department, why not? If I could get one into our back water ED 2 years ago, you can get one.

Personally I’m a C-Mac fan for ease of use and the ability to use it as a direct laryngoscope if there is too much blood and secretions down the hole to get a good video view.

Use a video laryngoscopy for every intubation so that everyone is familiar with it – rather than trying to get the hang of it when confronted with a difficult intubation. The whole team can see what is going on down the hole, I can see whether or not my colleague got the tube down the right hole.

That’s my 2c worth.

And a reminder to check that you are getting a good picture through the scope before you use it. Sometimes the techs don’t quite get all the goobies off the video lens – give it a wipe with an alcohol swab and you are back in action.

Emergency Medicine Tutorials

 

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Check the bladder size in old boys with UTIs

I was looking after an 80 year old man who had two weeks of recurrent fevers and rigors.  He had a confirmed UTI at the start of the illness: E.coli with standard sensitivities.  He had had 3 courses of appropriate antibiotics.

He'd come to ED the previous evening with a temp of 39.4?, hypotensive at 90/ and rigors.  He had been treated with gentamicin and then started on ciprofloxacin.  He had steadily improved but had had a further rigor at 0400.  When I saw him in the morning he was feeling much better and itching to go home.

He was tender in his L renal angle, afebrile and his urine dipstick was negative.  He had a moderately large belly but would have only been about 100kg but his bladder was hard to palpate but I thought it was dull half way to his umbilicus.

Bedside ultrasound showed a massive bladder up to his umbilicus.

He refused a catheter.  I chose to keep him a bit longer and sure enough he developed another fever at which time I convinced him to accept a catheter - which drained 3.5L urine.

I think the obstruction meant the kidneys just couldn't clear the infection.  I guess the dipstick was negative because the pus cells were just diluted in all that urine.

We left the catheter in pending urology outpatients input.


Check the bladder size in old boys with UTIs

I was looking after an 80 year old man who had two weeks of recurrent fevers and rigors.  He had a confirmed UTI at the start of the illness: E.coli with standard sensitivities.  He had had 3 courses of appropriate antibiotics.

He'd come to ED the previous evening with a temp of 39.4?, hypotensive at 90/ and rigors.  He had been treated with gentamicin and then started on ciprofloxacin.  He had steadily improved but had had a further rigor at 0400.  When I saw him in the morning he was feeling much better and itching to go home.

He was tender in his L renal angle, afebrile and his urine dipstick was negative.  He had a moderately large belly but would have only been about 100kg but his bladder was hard to palpate but I thought it was dull half way to his umbilicus.

Bedside ultrasound showed a massive bladder up to his umbilicus.

He refused a catheter.  I chose to keep him a bit longer and sure enough he developed another fever at which time I convinced him to accept a catheter - which drained 3.5L urine.

I think the obstruction meant the kidneys just couldn't clear the infection.  I guess the dipstick was negative because the pus cells were just diluted in all that urine.

We left the catheter in pending urology outpatients input.


Check the bladder size in old boys with UTIs

I was looking after an 80 year old man who had two weeks of recurrent fevers and rigors.  He had a confirmed UTI at the start of the illness: E.coli with standard sensitivities.  He had had 3 courses of appropriate antibiotics.

He'd come to ED the previous evening with a temp of 39.4?, hypotensive at 90/ and rigors.  He had been treated with gentamicin and then started on ciprofloxacin.  He had steadily improved but had had a further rigor at 0400.  When I saw him in the morning he was feeling much better and itching to go home.

He was tender in his L renal angle, afebrile and his urine dipstick was negative.  He had a moderately large belly but would have only been about 100kg but his bladder was hard to palpate but I thought it was dull half way to his umbilicus.

Bedside ultrasound showed a massive bladder up to his umbilicus.

He refused a catheter.  I chose to keep him a bit longer and sure enough he developed another fever at which time I convinced him to accept a catheter - which drained 3.5L urine.

I think the obstruction meant the kidneys just couldn't clear the infection.  I guess the dipstick was negative because the pus cells were just diluted in all that urine.

We left the catheter in pending urology outpatients input.


Privacy and Confidentiality

I recorded a talk on privacy and confidentiality by our hospital's lawyers for you - but the talk was too full of legal talk and section 2 of this Act, and this Code and "you have to work through these inclusion criteria, then these exclusion criteria considering all the necessary parts of the law, blah, blah, blah."  

Yeah right.

Key point is that we need to respect patients right to have their medical information remain confidential unless there is very good reason to disclose it.  The confidentiality of the doctor or nurse / patient relationship is considered very important in medicine and law.  Normally we need to have the patient's permission to disclose health information.  

We are free to share any relevant information with other health professionals involved in a patients care - unless the patient requests otherwise.  If the patient requests that we don't pass on information we must balance up the risks and benefits of not passing that information on.  It would be inappropriate to not pass on that the person was an infectious disease carrier to their surgeon who was about to operate.  

We may release the information without their permission if there is a credible specific, imminent and serious risk to the patient or others eg the patient says they are going to kill themselves or another named individual.  That information can only be divulged to someone who can do something about it eg the police and the potential victim.  The only information that is to be given is that information that will help to manage the risk. 


Generally anyone can know a patient is in hospital and what their condition is (not the disease but "critical", "stable" etc) unless there is reason to withhold that information eg a known assailant is looking for them.  

Another common example is breaking confidentiality to protect a child victim of abuse.  Generally we just have the child admitted and this avoids the problem for us.  Where there is suspected but not imminent risk we will report our concerns through our usual channels eg the hospital's child protection team.

Where a competent adult doesn't want us to report say partner assault injuries we are obliged to respect their request.  We are however free to report the injuries to another health professional for example a social worker who may not be bound by the same confidentiality rules as we are, and may be able to pass this information to the police.  This is what happens in practice in our hospital.  

We must release medical information if ordered by a search warrant or at the request of the police if they are acting on behalf of the coroner ie investigating a death.  There is usually no great urgency to release the information in these circumstances and this is usually best done in the cold light of day by the hospital's privacy staff.  They will ensure only the relevant information is released - eg making sure no health information about family members is released.  

When the police request information eg the details of a patient's injuries ideally we need the patient's permission to release this information.  We shouldn't assume that because they have reported the injury to the police that they give us permission to release their medical information. The police should request the information in writing and document the purpose of requesting the information is. The DHB generally releases information under rule 11 (2) (i) of the Health Information Privacy Code which requires agencies to uphold the maintenance of the law. Again this is best done in the cold light of day via the hospital's privacy team - tempting as it is to just give the information on the spot and get it over and done with.  

A couple of famous New Zealand cases are useful.  One is where a GP considered a school bus driver was not fit to drive.  He told members of the community and the local police officer.  The correct process was to request the patient agree not to drive.  If that wasn't successful the next step would be to go through the NZ Transport Agency.  The GP was told off by the medical council.  He then discussed the matter in the national media.  He was then struck off.

Another case that seems harsh at first glance is the case of a firearms license.  The police requested an opinion from a GP about the fitness of a patient to hold a firearms license.  The patient was known to have a very short fuse and had previously been abusive to practice staff.  The GP immediately replied to the police that the patient should not have been allowed a license.  He was told off by the medical council for this.  There was no imminent threat.  The police were not going to issue a license until they had heard from the GP.  The correct process would have been for the GP to have asked the patients permission to give that opinion.  If the patient refused the GP would then have had to weighed up the potential harm to the public from not telling the police vs the harm to the patient from breaking confidentiality.  The result would have been the same but the GP should have respected the patient's autonomy and discussed the matter with the patient first.

A case I didn't handle well involved a patient who came to ED after crashing a vehicle after taking a small overdose of sedatives.  He had a history of depression and was under the psych team as an outpatient.  He had a job with a lot of responsibility that involved public safety and driving vehicles at high speed.  He was seen by psych and assessed as fit for discharge.  One of the juniors in ED saw the patient.  He did not want his employer to know about this incident.  I didn't think this patient was safe to be on the road.  It was Sunday night of a long weekend.  I elected to speak to the patient's employer.  

The case did not end well.  In retrospect I should have asked the patient to agree not to drive.  If he refused to comply I should have then contacted the NZ Transport Agency.  I didn't have a right to talk to his employer. 

For children under 16 we should only disclose information to parents if the child lacks maturity or competence to make decisions for themselves and only if we believe the parent is acting in the child's best interests [Good Medical Practice.  New Zealand Medical Council]

A related matter is the confidentiality of work communications.  Anything you write or type during the course of your work, or even your recollection of a debriefing, is "discoverable" - you can be required to release it, or the hospital's IT department can be required to release it.  So be careful what you write down.  Sensitive communications are often better done face to face or by phone rather than by email.  Communications about colleagues health concerns should not be released without their permission, unless the public good outweighs their right to privacy. 

If you are unsure of the correct course of action please discuss with your colleagues or seek advice from your hospital's privacy officer (for Whanganui that is Trish Newton)


Thanks to Trish Newton for help with this item. 


Other references


Confidentiality and public safety.  Medical Council of New Zealand. 2002.  







Privacy and Confidentiality

I recorded a talk on privacy and confidentiality by our hospital's lawyers for you - but the talk was too full of legal talk and section 2 of this Act, and this Code and "you have to work through these inclusion criteria, then these exclusion criteria considering all the necessary parts of the law, blah, blah, blah."  

Yeah right.

Key point is that we need to respect patients right to have their medical information remain confidential unless there is very good reason to disclose it.  The confidentiality of the doctor or nurse / patient relationship is considered very important in medicine and law.  Normally we need to have the patient's permission to disclose health information.  

We are free to share any relevant information with other health professionals involved in a patients care - unless the patient requests otherwise.  If the patient requests that we don't pass on information we must balance up the risks and benefits of not passing that information on.  It would be inappropriate to not pass on that the person was an infectious disease carrier to their surgeon who was about to operate.  

We may release the information without their permission if there is a credible specific, imminent and serious risk to the patient or others eg the patient says they are going to kill themselves or another named individual.  That information can only be divulged to someone who can do something about it eg the police and the potential victim.  The only information that is to be given is that information that will help to manage the risk. 


Generally anyone can know a patient is in hospital and what their condition is (not the disease but "critical", "stable" etc) unless there is reason to withhold that information eg a known assailant is looking for them.  

Another common example is breaking confidentiality to protect a child victim of abuse.  Generally we just have the child admitted and this avoids the problem for us.  Where there is suspected but not imminent risk we will report our concerns through our usual channels eg the hospital's child protection team.

Where a competent adult doesn't want us to report say partner assault injuries we are obliged to respect their request.  We are however free to report the injuries to another health professional for example a social worker who may not be bound by the same confidentiality rules as we are, and may be able to pass this information to the police.  This is what happens in practice in our hospital.  

We must release medical information if ordered by a search warrant or at the request of the police if they are acting on behalf of the coroner ie investigating a death.  There is usually no great urgency to release the information in these circumstances and this is usually best done in the cold light of day by the hospital's privacy staff.  They will ensure only the relevant information is released - eg making sure no health information about family members is released.  

When the police request information eg the details of a patient's injuries ideally we need the patient's permission to release this information.  We shouldn't assume that because they have reported the injury to the police that they give us permission to release their medical information. The police should request the information in writing and document the purpose of requesting the information is. The DHB generally releases information under rule 11 (2) (i) of the Health Information Privacy Code which requires agencies to uphold the maintenance of the law. Again this is best done in the cold light of day via the hospital's privacy team - tempting as it is to just give the information on the spot and get it over and done with.  

A couple of famous New Zealand cases are useful.  One is where a GP considered a school bus driver was not fit to drive.  He told members of the community and the local police officer.  The correct process was to request the patient agree not to drive.  If that wasn't successful the next step would be to go through the NZ Transport Agency.  The GP was told off by the medical council.  He then discussed the matter in the national media.  He was then struck off.

Another case that seems harsh at first glance is the case of a firearms license.  The police requested an opinion from a GP about the fitness of a patient to hold a firearms license.  The patient was known to have a very short fuse and had previously been abusive to practice staff.  The GP immediately replied to the police that the patient should not have been allowed a license.  He was told off by the medical council for this.  There was no imminent threat.  The police were not going to issue a license until they had heard from the GP.  The correct process would have been for the GP to have asked the patients permission to give that opinion.  If the patient refused the GP would then have had to weighed up the potential harm to the public from not telling the police vs the harm to the patient from breaking confidentiality.  The result would have been the same but the GP should have respected the patient's autonomy and discussed the matter with the patient first.

A case I didn't handle well involved a patient who came to ED after crashing a vehicle after taking a small overdose of sedatives.  He had a history of depression and was under the psych team as an outpatient.  He had a job with a lot of responsibility that involved public safety and driving vehicles at high speed.  He was seen by psych and assessed as fit for discharge.  One of the juniors in ED saw the patient.  He did not want his employer to know about this incident.  I didn't think this patient was safe to be on the road.  It was Sunday night of a long weekend.  I elected to speak to the patient's employer.  

The case did not end well.  In retrospect I should have asked the patient to agree not to drive.  If he refused to comply I should have then contacted the NZ Transport Agency.  I didn't have a right to talk to his employer. 

For children under 16 we should only disclose information to parents if the child lacks maturity or competence to make decisions for themselves and only if we believe the parent is acting in the child's best interests [Good Medical Practice.  New Zealand Medical Council]

A related matter is the confidentiality of work communications.  Anything you write or type during the course of your work, or even your recollection of a debriefing, is "discoverable" - you can be required to release it, or the hospital's IT department can be required to release it.  So be careful what you write down.  Sensitive communications are often better done face to face or by phone rather than by email.  Communications about colleagues health concerns should not be released without their permission, unless the public good outweighs their right to privacy. 

If you are unsure of the correct course of action please discuss with your colleagues or seek advice from your hospital's privacy officer (for Whanganui that is Trish Newton)


Thanks to Trish Newton for help with this item. 


Other references


Confidentiality and public safety.  Medical Council of New Zealand. 2002.  







Privacy and Confidentiality

I recorded a talk on privacy and confidentiality by our hospital's lawyers for you - but the talk was too full of legal talk and section 2 of this Act, and this Code and "you have to work through these inclusion criteria, then these exclusion criteria considering all the necessary parts of the law, blah, blah, blah."  

Yeah right.

Key point is that we need to respect patients right to have their medical information remain confidential unless there is very good reason to disclose it.  The confidentiality of the doctor or nurse / patient relationship is considered very important in medicine and law.  Normally we need to have the patient's permission to disclose health information.  

We are free to share any relevant information with other health professionals involved in a patients care - unless the patient requests otherwise.  If the patient requests that we don't pass on information we must balance up the risks and benefits of not passing that information on.  It would be inappropriate to not pass on that the person was an infectious disease carrier to their surgeon who was about to operate.  

We may release the information without their permission if there is a credible specific, imminent and serious risk to the patient or others eg the patient says they are going to kill themselves or another named individual.  That information can only be divulged to someone who can do something about it eg the police and the potential victim.  The only information that is to be given is that information that will help to manage the risk. 


Generally anyone can know a patient is in hospital and what their condition is (not the disease but "critical", "stable" etc) unless there is reason to withhold that information eg a known assailant is looking for them.  

Another common example is breaking confidentiality to protect a child victim of abuse.  Generally we just have the child admitted and this avoids the problem for us.  Where there is suspected but not imminent risk we will report our concerns through our usual channels eg the hospital's child protection team.

Where a competent adult doesn't want us to report say partner assault injuries we are obliged to respect their request.  We are however free to report the injuries to another health professional for example a social worker who may not be bound by the same confidentiality rules as we are, and may be able to pass this information to the police.  This is what happens in practice in our hospital.  

We must release medical information if ordered by a search warrant or at the request of the police if they are acting on behalf of the coroner ie investigating a death.  There is usually no great urgency to release the information in these circumstances and this is usually best done in the cold light of day by the hospital's privacy staff.  They will ensure only the relevant information is released - eg making sure no health information about family members is released.  

When the police request information eg the details of a patient's injuries ideally we need the patient's permission to release this information.  We shouldn't assume that because they have reported the injury to the police that they give us permission to release their medical information. The police should request the information in writing and document the purpose of requesting the information is. The DHB generally releases information under rule 11 (2) (i) of the Health Information Privacy Code which requires agencies to uphold the maintenance of the law. Again this is best done in the cold light of day via the hospital's privacy team - tempting as it is to just give the information on the spot and get it over and done with.  

A couple of famous New Zealand cases are useful.  One is where a GP considered a school bus driver was not fit to drive.  He told members of the community and the local police officer.  The correct process was to request the patient agree not to drive.  If that wasn't successful the next step would be to go through the NZ Transport Agency.  The GP was told off by the medical council.  He then discussed the matter in the national media.  He was then struck off.

Another case that seems harsh at first glance is the case of a firearms license.  The police requested an opinion from a GP about the fitness of a patient to hold a firearms license.  The patient was known to have a very short fuse and had previously been abusive to practice staff.  The GP immediately replied to the police that the patient should not have been allowed a license.  He was told off by the medical council for this.  There was no imminent threat.  The police were not going to issue a license until they had heard from the GP.  The correct process would have been for the GP to have asked the patients permission to give that opinion.  If the patient refused the GP would then have had to weighed up the potential harm to the public from not telling the police vs the harm to the patient from breaking confidentiality.  The result would have been the same but the GP should have respected the patient's autonomy and discussed the matter with the patient first.

A case I didn't handle well involved a patient who came to ED after crashing a vehicle after taking a small overdose of sedatives.  He had a history of depression and was under the psych team as an outpatient.  He had a job with a lot of responsibility that involved public safety and driving vehicles at high speed.  He was seen by psych and assessed as fit for discharge.  One of the juniors in ED saw the patient.  He did not want his employer to know about this incident.  I didn't think this patient was safe to be on the road.  It was Sunday night of a long weekend.  I elected to speak to the patient's employer.  

The case did not end well.  In retrospect I should have asked the patient to agree not to drive.  If he refused to comply I should have then contacted the NZ Transport Agency.  I didn't have a right to talk to his employer. 

For children under 16 we should only disclose information to parents if the child lacks maturity or competence to make decisions for themselves and only if we believe the parent is acting in the child's best interests [Good Medical Practice.  New Zealand Medical Council]

A related matter is the confidentiality of work communications.  Anything you write or type during the course of your work, or even your recollection of a debriefing, is "discoverable" - you can be required to release it, or the hospital's IT department can be required to release it.  So be careful what you write down.  Sensitive communications are often better done face to face or by phone rather than by email.  Communications about colleagues health concerns should not be released without their permission, unless the public good outweighs their right to privacy. 

If you are unsure of the correct course of action please discuss with your colleagues or seek advice from your hospital's privacy officer (for Whanganui that is Trish Newton)


Thanks to Trish Newton for help with this item. 


Other references


Confidentiality and public safety.  Medical Council of New Zealand. 2002.  







A particularly dirty laceration.

A 58 year old farmer came to ED after crashing her quad bike.

She had a closed clavicle fracture which was treated with a haematoma block.

She had a 20cm linear laceration along her temporoparietal scalp with an arterial bleeder.


This was injected with long acting anaesthetic with adrenaline and a pressure dressing was put on it. We I went back half an hour later: no bleeding. We almost never have to tie off scalp bleeders.

Her parter turned up and said it must have been the grubber that had cut the scalp. A grubber is a tool for digging thistles and other weeds out of paddocks or fields.
"Oh" said the farmer, "I used that grubber to chop up a dead sheep yesterday"
"Why?"
"It was stuck by its legs in a bog and I couldn't pull the legs out, so I had to chop the legs off"

The wound received a particularly thorough scrub, high pressure irrigation. She received a tetanus booster and a dose of IV antibiotic (I used amoxicillin clavunate, which has good anaerobe cover - good for bugs in rotting sheep).

The wound was stapled closed.

A particularly dirty laceration.

A 58 year old farmer came to ED after crashing her quad bike.

She had a closed clavicle fracture which was treated with a haematoma block.

She had a 20cm linear laceration along her temporoparietal scalp with an arterial bleeder.


This was injected with long acting anaesthetic with adrenaline and a pressure dressing was put on it. We I went back half an hour later: no bleeding. We almost never have to tie off scalp bleeders.

Her parter turned up and said it must have been the grubber that had cut the scalp. A grubber is a tool for digging thistles and other weeds out of paddocks or fields.
"Oh" said the farmer, "I used that grubber to chop up a dead sheep yesterday"
"Why?"
"It was stuck by its legs in a bog and I couldn't pull the legs out, so I had to chop the legs off"

The wound received a particularly thorough scrub, high pressure irrigation. She received a tetanus booster and a dose of IV antibiotic (I used amoxicillin clavunate, which has good anaerobe cover - good for bugs in rotting sheep).

The wound was stapled closed.

A particularly dirty laceration.

A 58 year old farmer came to ED after crashing her quad bike.

She had a closed clavicle fracture which was treated with a haematoma block.

She had a 20cm linear laceration along her temporoparietal scalp with an arterial bleeder.


This was injected with long acting anaesthetic with adrenaline and a pressure dressing was put on it. We I went back half an hour later: no bleeding. We almost never have to tie off scalp bleeders.

Her parter turned up and said it must have been the grubber that had cut the scalp. A grubber is a tool for digging thistles and other weeds out of paddocks or fields.
"Oh" said the farmer, "I used that grubber to chop up a dead sheep yesterday"
"Why?"
"It was stuck by its legs in a bog and I couldn't pull the legs out, so I had to chop the legs off"

The wound received a particularly thorough scrub, high pressure irrigation. She received a tetanus booster and a dose of IV antibiotic (I used amoxicillin clavunate, which has good anaerobe cover - good for bugs in rotting sheep).

The wound was stapled closed.

? Record blood sugar

A patient with Type 1 diabetes ex IVDU on methadone, Hep C +ve, was BIBA with agitation, BSL "Hi"

The patient was agitated and resisting attempts at IV access - and had no veins.

Hmmm

Physical restraints, IN midazolam then 10mg haloperidol have no effect.
Given 250mg IM ketamine -> stopped wriggling -> femoral line.

Blood glucose 105mmol/L (N = 4 - 7.8) = 1892mg/dl = 18.9 g/L
pH 7.13
Na 123 (135 -147)
K 6.0 (3.4 - 5.2)
eGFR 17

Treated with IV normal saline at around 1 L normal saline over the first hour, then 500 mL over the second hour, then 500 mL 2-4 hourly thereafter, adjusted according to urine output and other clinical findings..  Insulin at about 0.5u/kg/hour.  Empiric broad spectrum antibiotics. 

Remained physically restrained, with opiate and propofol infusions for a day.  Steadily improved.

A question that came up was that he became hypernatraemic the next day and what should be done with that (not relevant for ED but you will need to know this for your medical and ICU runs.  

Many people, eg American Diabetic Association and UpToDate, use 1/2 normal saline for the hypernatraemic once the patient has been made euvolaemic (Peeing, JVP normal, mouth moist, HR and BP normalised).
The Christchurch Hospital Blue Book says half normal saline is seldom needed, and I've never used it for DKA.  As the Blue Book says the patient may become more hypernatraemic with treatment but their osmolarity will usually be falling appropriately.  And we don't want to change the patient's osmolarity too quickly as their hypertonic brain may absorb to much water causing cerebral oedema.  Well that's the theory anyway.

One way of finding his true Na is, in Australasian units:

Corrected Na = glucose (mmol/L) / 3.5 + measured Na
                      = 105 / 3.5 + 123
                      = 153

He is hypernatraemic.  DKAs can by hypo or hypernatraemic.  In this guys case the osmotic diuresis has sucked more water than sodium out of him.

On day 2 his measured Na was 153.  His glucose was 35.4.

Corrected Na = glucose / 3.5 + measured Na
                      = 35 / 3.5 + 153
                      = 163

His effective osmolarity  was:

2 x measured Na + glucose = 2 x 123 + 105
                                          = 351 on arrival

                                          = 2 x 153 + 35
                                          = 341 the next day. 

So his Na had gone up but his osmolarity is falling slowly, which is what we want.

The physicians here gave him 5% dextrose.  This has the potential to cause cerebral oedema as his brain is likely to still be hypertonic and would have sucked up the free water.  This may have contributed to his continued agitation. 

The other question is what to do about the raised K+ (6mmol/L) with his renal impairment.  DKA patients are always total body K+ depleted, even though there serum K+ may be high.  Insulin and normal saline will drop the serum K+.  So don't do anything else to drop the K and watch the K carefully.  As soon as the K normalises and he is peeing start replacing K+.  We can easily kill people by making them hypokalaemic in DKA. 

Emergency Medicine Tutorials


? Record blood sugar

A patient with Type 1 diabetes ex IVDU on methadone, Hep C +ve, was BIBA with agitation, BSL "Hi"

The patient was agitated and resisting attempts at IV access - and had no veins.

Hmmm

Physical restraints, IN midazolam then 10mg haloperidol have no effect.
Given 250mg IM ketamine -> stopped wriggling -> femoral line.

Blood glucose 105mmol/L (N = 4 - 7.8) = 1892mg/dl = 18.9 g/L
pH 7.13
Na 123 (135 -147)
K 6.0 (3.4 - 5.2)
eGFR 17

Treated with IV normal saline at around 1 L normal saline over the first hour, then 500 mL over the second hour, then 500 mL 2-4 hourly thereafter, adjusted according to urine output and other clinical findings..  Insulin at about 0.5u/kg/hour.  Empiric broad spectrum antibiotics. 

Remained physically restrained, with opiate and propofol infusions for a day.  Steadily improved.

A question that came up was that he became hypernatraemic the next day and what should be done with that (not relevant for ED but you will need to know this for your medical and ICU runs.  

Many people, eg American Diabetic Association and UpToDate, use 1/2 normal saline for the hypernatraemic once the patient has been made euvolaemic (Peeing, JVP normal, mouth moist, HR and BP normalised).
The Christchurch Hospital Blue Book says half normal saline is seldom needed, and I've never used it for DKA.  As the Blue Book says the patient may become more hypernatraemic with treatment but their osmolarity will usually be falling appropriately.  And we don't want to change the patient's osmolarity too quickly as their hypertonic brain may absorb to much water causing cerebral oedema.  Well that's the theory anyway.

One way of finding his true Na is, in Australasian units:

Corrected Na = glucose (mmol/L) / 3.5 + measured Na
                      = 105 / 3.5 + 123
                      = 153

He is hypernatraemic.  DKAs can by hypo or hypernatraemic.  In this guys case the osmotic diuresis has sucked more water than sodium out of him.

On day 2 his measured Na was 153.  His glucose was 35.4.

Corrected Na = glucose / 3.5 + measured Na
                      = 35 / 3.5 + 153
                      = 163

His effective osmolarity  was:

2 x measured Na + glucose = 2 x 123 + 105
                                          = 351 on arrival

                                          = 2 x 153 + 35
                                          = 341 the next day. 

So his Na had gone up but his osmolarity is falling slowly, which is what we want.

The physicians here gave him 5% dextrose.  This has the potential to cause cerebral oedema as his brain is likely to still be hypertonic and would have sucked up the free water.  This may have contributed to his continued agitation. 

The other question is what to do about the raised K+ (6mmol/L) with his renal impairment.  DKA patients are always total body K+ depleted, even though there serum K+ may be high.  Insulin and normal saline will drop the serum K+.  So don't do anything else to drop the K and watch the K carefully.  As soon as the K normalises and he is peeing start replacing K+.  We can easily kill people by making them hypokalaemic in DKA. 


Emergency Medicine Tutorials


? Record blood sugar

A patient with Type 1 diabetes ex IVDU on methadone, Hep C +ve, was BIBA with agitation, BSL "Hi"

The patient was agitated and resisting attempts at IV access - and had no veins.

Hmmm

Physical restraints, IN midazolam then 10mg haloperidol have no effect.
Given 250mg IM ketamine -> stopped wriggling -> femoral line.

Blood glucose 105mmol/L (N = 4 - 7.8) = 1892mg/dl = 18.9 g/L
pH 7.13
Na 123 (135 -147)
K 6.0 (3.4 - 5.2)
eGFR 17

Treated with IV normal saline at around 1 L normal saline over the first hour, then 500 mL over the second hour, then 500 mL 2-4 hourly thereafter, adjusted according to urine output and other clinical findings..  Insulin at about 0.5u/kg/hour.  Empiric broad spectrum antibiotics. 

Remained physically restrained, with opiate and propofol infusions for a day.  Steadily improved.

A question that came up was that he became hypernatraemic the next day and what should be done with that (not relevant for ED but you will need to know this for your medical and ICU runs.  

Many people, eg American Diabetic Association and UpToDate, use 1/2 normal saline for the hypernatraemic once the patient has been made euvolaemic (Peeing, JVP normal, mouth moist, HR and BP normalised).
The Christchurch Hospital Blue Book says half normal saline is seldom needed, and I've never used it for DKA.  As the Blue Book says the patient may become more hypernatraemic with treatment but their osmolarity will usually be falling appropriately.  And we don't want to change the patient's osmolarity too quickly as their hypertonic brain may absorb to much water causing cerebral oedema.  Well that's the theory anyway.

One way of finding his true Na is, in Australasian units:

Corrected Na = glucose (mmol/L) / 3.5 + measured Na
                      = 105 / 3.5 + 123
                      = 153

He is hypernatraemic.  DKAs can by hypo or hypernatraemic.  In this guys case the osmotic diuresis has sucked more water than sodium out of him.

On day 2 his measured Na was 153.  His glucose was 35.4.

Corrected Na = glucose / 3.5 + measured Na
                      = 35 / 3.5 + 153
                      = 163

His effective osmolarity  was:

2 x measured Na + glucose = 2 x 123 + 105
                                          = 351 on arrival

                                          = 2 x 153 + 35
                                          = 341 the next day. 

So his Na had gone up but his osmolarity is falling slowly, which is what we want.

The physicians here gave him 5% dextrose.  This has the potential to cause cerebral oedema as his brain is likely to still be hypertonic and would have sucked up the free water.  This may have contributed to his continued agitation. 

The other question is what to do about the raised K+ (6mmol/L) with his renal impairment.  DKA patients are always total body K+ depleted, even though there serum K+ may be high.  Insulin and normal saline will drop the serum K+.  So don't do anything else to drop the K and watch the K carefully.  As soon as the K normalises and he is peeing start replacing K+.  We can easily kill people by making them hypokalaemic in DKA. 

Emergency Medicine Tutorials


Phenylephrine refresher

Just a quick reminder about phenylephrine.

Phenylephrine is a pure alpha agonist = pure vasoconstrictor.  So it causes peripheral arterioles and venules to constrict with out any ß effect.  So you get a rise in BP with no tachycardia - you may get a reflex bradycardia. 

Great for the hypotensive AF patient.  For the patient with AF, hypotension and pulmonary oedema - who knows?  Seems to keep them alive. 

It's also great for antagonising the hypotensive effects of propofol and other anaesthethics.  Also a good short term pressor while you are getting say your noradrenaline / norepinephrine infusion made up.

Take a 10mg vial.  Add its contents to 100ml of normal saline.  Shake.  Draw off 10ml of the resultant solution.  = 100µg/ml.   Label the bag and the syringe. 

Typical dose 2ml = 200µg will give about 5 minutes of vasoconstriction.

Phenylephrine refresher

Just a quick reminder about phenylephrine.

Phenylephrine is a pure alpha agonist = pure vasoconstrictor.  So it causes peripheral arterioles and venules to constrict with out any ß effect.  So you get a rise in BP with no tachycardia - you may get a reflex bradycardia. 

Great for the hypotensive AF patient.  For the patient with AF, hypotension and pulmonary oedema - who knows?  Seems to keep them alive. 

It's also great for antagonising the hypotensive effects of propofol and other anaesthethics.  Also a good short term pressor while you are getting say your noradrenaline / norepinephrine infusion made up.

Take a 10mg vial.  Add its contents to 100ml of normal saline.  Shake.  Draw off 10ml of the resultant solution.  = 100µg/ml.   Label the bag and the syringe. 

Typical dose 2ml = 200µg will give about 5 minutes of vasoconstriction.

Phenylephrine refresher

Just a quick reminder about phenylephrine.

Phenylephrine is a pure alpha agonist = pure vasoconstrictor.  So it causes peripheral arterioles and venules to constrict with out any ß effect.  So you get a rise in BP with no tachycardia - you may get a reflex bradycardia. 

Great for the hypotensive AF patient.  For the patient with AF, hypotension and pulmonary oedema - who knows?  Seems to keep them alive. 

It's also great for antagonising the hypotensive effects of propofol and other anaesthethics.  Also a good short term pressor while you are getting say your noradrenaline / norepinephrine infusion made up.

Take a 10mg vial.  Add its contents to 100ml of normal saline.  Shake.  Draw off 10ml of the resultant solution.  = 100µg/ml.   Label the bag and the syringe. 

Typical dose 2ml = 200µg will give about 5 minutes of vasoconstriction.

Salvage cardioversion of a surgical patient

A fun case.

We received word that ICU/CCU was full and a surgical registrar wanted to bring a patient to ED from the surgical ward to give adenosine for an SVT.

ED was quiet so I wandered up to the ward for a nosey.

The patient was 86 and 3 days post a hemicolectomy.  At 9pm her obs had been fine but 1/2 an hour later she had suddenly become SOB and faint. 

The surgical reg showed me the ECG,  fast AF, rate 160, with some lateral ST depression.
"AF" I said.
"Is it?" said the surgical reg.
"Yes" said the first year house officer 3 months out of med school.

No Hx of AF.  No Hx of CCF
The patient was short of breath, Sats 97% on high flow O2, fine creps both bases.  Pale, clammy, JVP to earlobes, BP 87/50, HS dual + nil, calves soft, no oedema, reluctant to talk but orientated, afebrile


Hmmm septic, PE, spontaneous AF, MI?  Probably not septic given sudden onset.

Transferred to ED

Phenylephrine 200mcg (Does it help?  Don't know but I want a half decent MAP to hopefully maintain coronary perfusion under anaesthetic)
Fentanyl 50mcg
Etomidate 5mg

200J -> SR for 2 minutes then AF
200J -> SR for 2 minutes then AF

Amiodarone 150mg push
BP drops
Phenylephrine 200mcg

200J -> SR for 2 minutes then AF

Crap

Amiodarone 150mg push
BP drops
Phenylephrine 200mcg

Fentanyl 25mcg
BP drops
Phenylephrine 200mcg

Etomidate 5mg

200J -> SR and stays in SR.  ST segments normal.

K+ a little low -> K, mag and amiodarone infusions (luckily patient still had a central line in)

Mild chest discomfort where the pads are.  No SOB, chest clear.

Tropinin before electricity normal - still could be an early MI.
PE unlikely given resolution of symptoms.  

Patient did well. 





Salvage cardioversion of a surgical patient

A fun case.

We received word that ICU/CCU was full and a surgical registrar wanted to bring a patient to ED from the surgical ward to give adenosine for an SVT.

ED was quiet so I wandered up to the ward for a nosey.

The patient was 86 and 3 days post a hemicolectomy.  At 9pm her obs had been fine but 1/2 an hour later she had suddenly become SOB and faint. 

The surgical reg showed me the ECG,  fast AF, rate 160, with some lateral ST depression.
"AF" I said.
"Is it?" said the surgical reg.
"Yes" said the first year house officer 3 months out of med school.

No Hx of AF.  No Hx of CCF
The patient was short of breath, Sats 97% on high flow O2, fine creps both bases.  Pale, clammy, JVP to earlobes, BP 87/50, HS dual + nil, calves soft, no oedema, reluctant to talk but orientated, afebrile


Hmmm septic, PE, spontaneous AF, MI?  Probably not septic given sudden onset.

Transferred to ED

Phenylephrine 200mcg (Does it help?  Don't know but I want a half decent MAP to hopefully maintain coronary perfusion under anaesthetic)
Fentanyl 50mcg
Etomidate 5mg

200J -> SR for 2 minutes then AF
200J -> SR for 2 minutes then AF

Amiodarone 150mg push
BP drops
Phenylephrine 200mcg

200J -> SR for 2 minutes then AF

Crap

Amiodarone 150mg push
BP drops
Phenylephrine 200mcg

Fentanyl 25mcg
BP drops
Phenylephrine 200mcg

Etomidate 5mg

200J -> SR and stays in SR.  ST segments normal.

K+ a little low -> K, mag and amiodarone infusions (luckily patient still had a central line in)

Mild chest discomfort where the pads are.  No SOB, chest clear.

Tropinin before electricity normal - still could be an early MI.
PE unlikely given resolution of symptoms.  

Patient did well. 





Salvage cardioversion of a surgical patient

A fun case.

We received word that ICU/CCU was full and a surgical registrar wanted to bring a patient to ED from the surgical ward to give adenosine for an SVT.

ED was quiet so I wandered up to the ward for a nosey.

The patient was 86 and 3 days post a hemicolectomy.  At 9pm her obs had been fine but 1/2 an hour later she had suddenly become SOB and faint. 

The surgical reg showed me the ECG,  fast AF, rate 160, with some lateral ST depression.
"AF" I said.
"Is it?" said the surgical reg.
"Yes" said the first year house officer 3 months out of med school.

No Hx of AF.  No Hx of CCF
The patient was short of breath, Sats 97% on high flow O2, fine creps both bases.  Pale, clammy, JVP to earlobes, BP 87/50, HS dual + nil, calves soft, no oedema, reluctant to talk but orientated, afebrile


Hmmm septic, PE, spontaneous AF, MI?  Probably not septic given sudden onset.

Transferred to ED

Phenylephrine 200mcg (Does it help?  Don't know but I want a half decent MAP to hopefully maintain coronary perfusion under anaesthetic)
Fentanyl 50mcg
Etomidate 5mg

200J -> SR for 2 minutes then AF
200J -> SR for 2 minutes then AF

Amiodarone 150mg push
BP drops
Phenylephrine 200mcg

200J -> SR for 2 minutes then AF

Crap

Amiodarone 150mg push
BP drops
Phenylephrine 200mcg

Fentanyl 25mcg
BP drops
Phenylephrine 200mcg

Etomidate 5mg

200J -> SR and stays in SR.  ST segments normal.

K+ a little low -> K, mag and amiodarone infusions (luckily patient still had a central line in)

Mild chest discomfort where the pads are.  No SOB, chest clear.

Tropinin before electricity normal - still could be an early MI.
PE unlikely given resolution of symptoms.  

Patient did well.