Flanno’s FOAM findings #5

I've been sayin it for years... well, close to a year now:

we need to stop teaching and expecting our medical students, junior doctors and 'real world' practicing doctors to be experts in critical appraisal of EBM, let the experts be experts in appraised EBM.  Instead, we need to teach our underlings (and ourselves) how to find that expert apprasial and take it to the bedside (knowledge translation).   Now it seems there is some evidence and other smart people who are thinking the same way:

learning information management not EBM

 

DRAFT AIRWAY PATHWAY

G'day all,

In reponse to all the recent online talk of DSI vs RSI and DL vs VL, I have modified my airway algorithm. Basically I'm not ready to give up on DL completely, and am an enthusiastic adopter of high flow nasal O2, which has already bailed me out of a few hairy situations. This is just a draft, and I'm looking for feedback... Check out the links below for the background material out in the ED ether

 

 

http://prehospitalmed.com/2013/01/26/delayed-sequence-intubation-or-dci-deadly-critical-intubation-nope-that-doesnt-sound-better/ 

http://i1.wp.com/emcrit.org/wp-content/uploads/2013/02/VortexCognitiveAid.png

 http://mdaware.blogspot.com.au/2013/03/rsa-ok.html

 http://emcrit.org/podcasts/emcrit-intubation-checklist/?utm_source=feedburner&utm_medium=feed&utm_campaign=Feed%3A+emcrit+%28EMCrit+Blog+-+Emergency+Critical+Care%29&utm_content=Google+Reader

 http://prehospitalmed.com/2013/04/19/if-you-had-45-minutes-could-you-intubate-anyone/

regrets.

after the obvious hacking that occured over the weekend, I have had to place the educator's blog behind our password protected firewall, regretfully.  It is probably safer this way...

Flanno’s FOAM findings #4

FOAM and Fellowship study intersect…

studying for fellowship, found the previous SAQ below:

1. Discuss the evidence regarding the use of Oxygen therapy in the Emergency

Department in relation to the following presentations. (100%)

a. Acute coronary syndromes

b. Acute exacerbations of chronic airways limitation (cf. CAL / COPD)

what the what???  I found this one rather difficult until I serendipitously came across these FOAM posts below

RESUS.me

Scancrit.org

underneathem.org

UMEM educational pearls

emergencyeducation.net.

ANSWER:

discuss = pro/con, list

Key Issues:

  • oxygen therapy is cornerstone of treatment modality in critically unwell patient
  • not evidence based
  • essesntial in hypoxic patients (O2 sat<90%) and in cardiac arrest, Pre-ox for RSI, sepsis, shock, trauma, near-drowning, CO poisoning
  • evidence suggest ‘supra-normal’ levels detrimental
  • best practice is to titrate oxygen therapy to a peripheral 02 saturations between 88-94%

oxygen in ACS:

pro:

  • vital in hypoxic patient to ensure optimal oxygen delivery to tissue, namely: myocardium,
  • simple to apply,
  • cheap,
  • recently defined as standard of care

con:

  • emerging evidence suggests supra-normal levels of oxygenation has potential harm in not only ACS, but neonatal resuscitation, COPD, post cardiac arrest, Stoke, TBI, and ARDS.
  • Namely through production of toxic free radicals, excessive oxidative stress to reperfused tissues, widespread vasoconstriction of coronary and cerebral vessels, and associated increase SVR, potential increase V/Q mismatch
  • most recent AHA/ACC guidelines de-empasize High-flow use of oxygen in uncomplicated MI/ACS patients, suggesting rather a titrated dosage, aiming for FiO2~40%

**might be even more clever to comment that more answers to come with the publication of the AVOID trial (A randomized controlled trial of oxygen therapy in acute myocardial infarction Air Verses Oxygen In myocarDial infarction)

Oxygen in COPD

Pro:

  • vital in the critically hypoxic patient 
  • simple to apply 
  • cheap  
  • ubiquitous in all facets of health care provision 

con:

  • patients prone to retain CO2 due to Chronic airway limited disease processes have reset central chemoreceptors that normally stimulate breathing in response to dec. pH/inc CO2, this results in reliance on peripheral receptors responding to hypoxic to stimulate respiration.  high arterial O2=no stimulus to breath=continued rise in CO2=acidosis,deterorating LOC. 
  • similar to systemic effects described for ACS: oxidative stress, free radicals, vasoconstriction, V/Q mismatch. 

 **I assume buffing this part of the answer with a comment on a recent Hobart pre-hospital study showing increased mortality in COPD patient receiving high flow O2 is going to make you look good.

Boo-yah!!  nailed it,  NO Cameron/tintinalli/Dunn needed, thank you very much. VIVA la FOAMed….

PS: I would like to see what an answer like that would score… I pointing at you Mark  and recent fellowshites!  as well as anyone else who might have some good suggestions for a better answer: I found it very diffcult to come up with ‘pros’  seems obvious doesn’t, anybody have others?

 

US guided nerve blocks

Want to learn how to do femoral/sciatic/tibial/radial/ulna/median nerve blocks as well as shoulder reductions under local anaesthetic?!

Check out this 45 minute lecture by Mike Stone of US podcast fame.

Enjoy

http://vimeo.com/31010728

what sedative should I use?

further to our busy weekend, Saira and I had discussion about appropriate sedation in TBI. This is actually the wrong question to be focusing on as you will see below, it does not matter, just use something. What does matter are the other components to good secondary brain injury prevention:

basics in head trauma:

  • let gravity help with pressure (head of bed up, no constricting noose around neck to obstruct venous flow i.e. collar, ETT tape)
  • shut their brain off (dec. metabolic demand)
  • keep enough Blood to the brain (CPP = MAP-ICP)  ; need a MAP >80, this may be a caution with propofol
  • keep everything in blood normal:CO2,BSL,O2,temperature.

I cant find anywhere to suggest inc. in ICP
http://lifeinthefastlane.com/book/critical-care-drugs/propofol/

some protocols favor it in traumatic head injury
http://emcrit.org/podcasts/high-icp-herniation/

in reality, it does not matter what sedative you use, just use something as that is defnitely know to worsen ICP and outcomes 
http://resus.me/sedation-for-traumatic-brain-injury/
http://www.ncbi.nlm.nih.gov/pubmed/22094498

Anyone disagree?

scott

 

 

Flanno’s FOAM findings #3

  

 

Busy times at the ol’ Maitland ED this past weekend, unsuspecting adolescent head traumas, old guys with septic shoulders, uncontrollable hepatic encephalopathies, rugby players with neck fractures and neurologic deficits and, a 2yo with mid-shaft fractured femur…   lots of FOAMy goodness here, lots I could report on how the use of blogs, podcasts, and social media helped us manage the madness.  After re-telling the stories of the weekend to a few sorry souls who had to listen, I was a little taken aback by the quizzical looks, follow up questions, and interest in how to do femoral nerve blocks for the acute treatment of mid-shaft femur fractures, even in 2yo’s. So, topic picked!

Disclaimer: not here to re-invent the wheel, only direct you to the online resources that have.

Femoral nerve blocks work:

http://journals.lww.com/pec-online/Abstract/2012/02000/Ultrasound_Guided_Femoral_Nerve_Block_for_Pain.21.aspx

http://www.annemergmed.com/article/S0196-0644(06)02261-X/abstract

even the orthopaedic guys think so:

http://jbjs.org/article.aspx?articleid=27863

it is easy to learn online:

http://www.neuraxiom.com/html/newfemoral.html

http://www.neuraxiom.com/html/ficb.html

Silly not to do it with USS

http://www.ultrasoundpodcast.com/2012/03/episode-24-femoral-nerve/

http://www.ultrasoundvillage.com/imagelibrary/cases/?id=121&media=456&testyourself=0

http://www.sonoguide.com/femoral_nerve_block.html

great tip on dosing: 

mix up a 50:50 solution of 1% lidocaine with 0.5% bupivacaine and, before ultrasound guidance, used 1mL per year of age. It works out as less than 2mg/kg of lidocaine (usually) and less than 1mg/kg bupivacaine

http://stemlynsblog.org/2012/07/through-the-looking-glass-chirocaine-vs-bupivicaine/

its in our exams;

http://lifeinthefastlane.com/2009/11/quiz-paediatrics-013/

bottom line, just learn to do it, your patients will thank you.

scott

 

 

guidelines for me…

amalgamating the worlds guidelines relevant to Emergency Medicine in a very searchable fashion...

I have added it to our 'on the floor' reference page. check it out. organized and updated by Tessa Davis, Paediatric EM trainee originally from Glasgow now living in Sydney.

be great if we could get HNEH guidelines up there, yea, like that is going to happen..... Dafters, you still looking for non-clinical projects, I know of a few...


Intubation checklist

Just listened to a very thought provoking podcast by Scott Weingart on developing a checklist for intubation.

Checklists have previously found to be very helpful in medical procedures (eg decreasing central line infection rates), and I think we've all seen/made errors in high stress situations like RSI.

Attached below is the link.

http://emcrit.org/podcasts/emcrit-intubation-checklist/

Is this something we should be adding to our list of protocols?

SMACC2013

As you may know, SMACC 2013 just finished. Scott, myself and a few other HNE (mainly ED) people were there - a good turn out. 

 

The conference was awesome. If you don't yet believe that FOAM, social media and the repository of critical care experience that it represents is worth reading, keep an eye on the SMACC2013 website - the talks will be released there in the next week or so. Free, and open access. 

Highlights were too numerous to mention. 

Flano’s FOAM findings #2

  

Continuing on with the anecdotal evidence of FOAM resources improving (at least changing) my day to day clincal work:

I have a love/hate relationship with paediatric febrile presentations:  on one hand, the majority of them are benign and a little parental reassurance goes a long way. On the other hand, they can be the harbinger of very morbid disease process and our paediatric colleagues continue to seem obsessed with toddler wee which can be quite frustrating (and seemingly worthless) to obtain sometimes..... 

Here are some recent blogs/articles/tips to add confidence to your assessment and quicken the dispostion of this large group of patients in our departments:

  • risk of serious bacterial infection is VERY low in the well looking, immunized child:

http://embasic.org/2013/02/25/essential-evidence-7-saem-occult-bacteremia/

____________________________________________________________________________________

  • tympanic temperature recording is proven to be both specific and sensitive, dont need to stick things up kids bums and it was better then under the arm. Interestingly, the infrared recording of the temporal artery was also very sensitive/specific (I previously thought that was all just witchcraft..)

http://pemlit.org/2013/01/04/4th-january-2013-comparison-of-rectal-axillary-tympanic-and-temporal-artery-thermometry-in-the-pediatric-er/

____________________________________________________________________________________

  • great tip on getting a quick wee from those non-compliant little toddlers, boys only, sorry girls, still difficult. (jeez, learn to pee on demand dam it!!)

http://www.impactednurse.com/?p=5485

In my N=4, I have not had to proceed past step 2, so be ready

__________________________________________________________________________________

  • Procalcitonin is fraught with tainted evidence and conflicts of interest.... I choose to ignore anyone who wants this test at the moment... (provided it is not my paediatrician wife who I am bound to obey.... silly vows!)

http://www.emlitofnote.com/2013/02/jama-procalcitonin.html

http://www.emlitofnote.com/2013/02/jama-integrity-accessibility-and-social.html

__________________________________________________________________________________

  • Why is the urine sample so important:

Healthy, normal children (3-36month) do not progress to sepsis, <1% of all serious bacteria infections

renal scarring/CKD post recurring febrile UTI is controversal at best.

http://empem.org/2012/12/pediatric-uti-controversies/

http://www.smartem.org/podcasts/pediatric-uti-its-about-future

http://pediatrics.aappublications.org/content/early/2011/10/06/peds.2010-3520.abstract

___________________________________________________________________________________

  • How my practice has changed: risk stratify, maybe urine sample, maybe treat, always close follow up.

http://www.epmonthly.com/subspecialties/pediatrics/aap-issues-new-uti-guidelines/

http://www.nice.org.uk/nicemedia/live/11819/36030/36030.pdf

___________________________________________________________________________________ 

And as per the last post, I promise you I have done no extra work outside of my regular persual of FOAMed, I have read none of the publications in full on this topic, that I can promise you. (I have no business attmepting to critically appraise the literature, leave it to the evolving number of experts out there.)

drink it up. Scott

 

 

Free-Open-AMed

pending any objections over the course of this week, monday, march 11th, in honor of the start of the SMACC conference, the educator's blog with no longer be behind the hneed.com password protection area.   

 

**please note that once this happens, any and all posts will be at the responsiblity of the author and in accordance with the widely available disclaimer for the hneed.com site. Please review this to ensure you are in agreeance.

Question regarding absorbable sutures in kids

An important question was raised a couple of days ago regarding the use of absorbable sutures on kids faces. We usually advise removal of non absorbable facial sutures at 3 days to prevent scarring due to epithelialisation of the suture tracts which can occur from the 5 day mark onwards. I note that some of the absorbable sutures can take longer than this to dissolve. The Karounis paper does suggest good cosmetic results but I note that it was a study with considerable refusals and losses to follow up so potentially a degree of bias. Should we be following kids with absorbable facial sutures up at 5 days and removing them anyway if there is suture material left?

comment please.

Given that the F of FOAM stands for free and the O for open, I have been contemplating making 'the educators blog'  free and open to the medical community at large (i.e. placing it outside the password protection), please comment below to encourage or dissuade me over the next 2 weeks, after that, I will have attended the SMACC conference and drank more of the FOAM juice and may not be turned back!!! 

 

Flanno’s FOAM Findings #1

A couple of weeks ago I promised to display how my continued review of websites/blogs/podcasts improved my clinical knowledge almost on a day to day basis. Regretfully, it has been more then a couple of weeks since that declaration, but I am going to start right here: 

 

late last week I was asked to discuss a case at handover involving a patient presenting SOB, hypoxic, tachycardic and hypotensive with a background of metastatic cancer and multiple previous DVT/PE.  Interestingly, this patient had an INR of 3.0 and platelets of 40....  the discussion revolved around PE diagnoses', differentiating 'submassive' from 'massive' PE and the indicators of when to thrombolyse, as well as discussion about a recently published study known as 'the MOPETT study' suggesting half dose thrombolysis in the groups currently not recommended for thrombolysis....

I was able to actively participate in the discussion solely because over the previous couple of weeks, I had come across these posts. There was no active searching, no extra reading, no extra time spent on my part specifically looking into evidence and treatment of pulmonary embolus. Just simply reading/listening to/watching 10 posts a day, taking a total of 45min, sometimes on the shitter, sometimes while in the car, and sometimes while watching The Block: Allstars with my wife!:

http://emlyceum.com/2013/02/01/pulmonary-embolism-answers/

http://keepingup.vanderbiltem.com/images/podcast/keeping_up_update_v114.mp3

http://www.emlitofnote.com/2013/01/mopett-half-dose-tpa-for-pe.html

http://emcrit.org/wee/mopett-trial/

http://pulmccm.org/2013/cardiovascular-disease-review/forget-embolic-burden-of-pulmonary-embolism-location-is-everything-chest/

Let the FOAM float to the top. cheers.

 

 

Home cricothyrotomy trainer

Just found a new blog linked to on weingarts emcrit site. 

It's called SOCMOB (short for standing on the corner minding my own business)

and it has a great post on a home made cric trainer. 

Well worth a look

http://socmob.blogspot.com.au/2013/01/how-to-make-your-own-cricothyrotomy.html 

How to get the FOAM to go down nice and smooth

As discussed at our teaching session yesterday.  I am including a simple ‘how to’ test the waters of Social media, FOAMed, and the internet resources that are, in my mind, invaluable to the future of Emergency Medical education.  

Benefits of the FOAMed revolution:

 

  1. Keeps you fresh and up-to-date on relevant literature and topics in the world of Emergency medicine/critical care with suprisingly little effort once you have setup the appropriate applications and programs necessary on the various computing devices
  2. syncs well with adult learning and the concept of asynchronous learning.  Allows us, as adults with varying backgrounds and knowledge, busy with rotating shift times, family commitments, and extracurricular activities to filter in what we individually need to know, filter out what we do not and access it at our own pace, leisure, time
  3. Cuts the Knowledge translation time down from the quoted 10 years to, often, less then 1. ( i.e. time it takes for good evidence based therapy to reach the bedside/patient care.)
  4. Adds context to the overwhelming amount of information we, as emergency physicians, are supposed to know.  (i.e.  reading a chapter in tintinalli on cardiogenic shock will take you through all of the potential causes, therapies and complications but reading/listening to a self-selected group of 4-5 different blogging emergency physicians about what is realistically, clinically important with cardiogenic shock adds a context to the subject about what is indeed common, relevant and important and what is simply, examinable material.)
  5. Has potential to connect you with amazing clinicans/educators around the world with multiple years of experience and individual expertise without the ‘phobia’ that may have prevented you from asking that question while in the audience or walking up to meet and clarify a point made during a presentation by an 'expert in the field' when you were at that last conference.   Twitter has truly revolutized communication between the student and the educator.  Quick, well thought out questions are quickly and succinctly answered from anywhere in the world, often with multiple opinions, regardless of where you sit in the hierarchy of medical education/practice.
  6. Allows our hardwork and preparation of content to NOT go unnoticed when the discussion topic you vigorously prepared for a group of 10 ended up being 2.  put it up into the FOAM for all to see.

Drawbacks:  

I personally seen none. 

 **I do see a negative effect of reliance on smartphone apps and search engines at the bedside in time critical situations. We do need to maintain a core base of knowledge in our RAM (random access memory) but perhaps that ‘core’ knowledge is changing and time critical thinking would be better served by use of well reviewed, rehearsed checklists such as this, or this, or this (possibly found on that very smartphone?).

 

 NOW, How to drink the FOAM without coughing it back up  (from my perspective)

 

  1. sign up to a ‘feed aggregator’  This is a computer program or phone application that allows you to subscribe to particular websites that you would like to routinely check for updated information.  Anytime any of those particular websites produce new content, you will be notified by you feed aggregator.  Thereby making if effortless to check upward of 90 websites a day (if you are so inclined)
    • popular examples:  google readerfeedly
    • tip:  it is best to use google reader as the backbone interface so that all of your different mobile devices can routinely sync with it (meaning if you read something on your iphone, the next time you look at your ipad, it will come up as already 'read')
  2. chose the sites to follow:
    • start simple with www.lifeinthefastlane.com, follow their  LITFL review each week for the best summary of recent online activity.
    • check www.emgoogle.com which searches all of the known Emergency medicine/critical care theme websites/blogs/podcasts.  On this site you will find a number of links to sites that are and have attempted to collate the massively expanding list of high quality free educational resources: I do not recommend you sift through website after website as you will get frustrated and feel like it is too much.  As stated previously, use a filter and let time determine where you are finding the most useful information relevant to you
    • HERE are the websites and blogs that I currently follow.
  3. STRICTLY limit yourself to a certain amount of content/time period
  4. sign up for a twitter account.  a simple way to ‘experience’ twitter for Emergency medicine education purposes is to ‘discover’ the hashtag #FOAMed.  you will understand once you have signed in to twitter,  a hashtag simply means that when an someone makes a 'tweet' and includes #....  everyone whole is following this #... will be notified of their post. It is quite useful even if you do not interact and just observe.  There are questions of the day, xrays of the week, simple questions we ask ourselves everyday with multiple sourced answers….
  5. Relax, let the filter (you can’t go wrong by following what LITFL suggest) sort through the noise and let time tell you what it is you want from this amazing tool

I have unknowingly become a zealot for the use of social media, web-based education and all things FOAM, by accident really, it truly was not my intention. However, I not only believe in its value but, almost on a daily basis, personally benefit from it.  I am more then happy to discuss, show, give tips/suggestions on how to navigate it all without losing days of your life.  

In fact, I am going to attempt to start a semi-regular post on this blog (weekly maybe) detailing one example of how FOAM has improved my background/foreground knowledge in some way…. think I will call it Flano’s FOAM findings, or something cheesy like that, any suggestions?

cheers.scott

 

GCS 8 = Intubate?

Currently at the Mater, so some Tox related stuff.....

The GCS was described and validated for prognostication in head trauma. What about if you are drugged up?

If a patient is intoxicated and head trauma, which is which?

This study (http://www.ncbi.nlm.nih.gov/pubmed/17159670) in the Journal of Trauma 2006, showed no significant change in GCS for blunt head injured patients due to their intoxication. Read the study for full details, but basically - if the GCS is low, it's because of the brain injury not the booze!

 

If a patient is intoxicated with no head trauma:

This study (http://www.ncbi.nlm.nih.gov/pubmed/19546273) in the EMJ 2009 (n=26), and this study (http://www.ncbi.nlm.nih.gov/pubmed/19272743) in the Journal of Emerg Med 2009 (n=73) might help you decide.

Both studies looked at poisoned/intoxicated patients in a prospective observational manner. Findings for the EMJ study were that clinical assesment of airway patency and protection (clinician's gestalt) were as good as GCS in determining need for intubation. The second larger study observed poisoned patients with no intubation. 12 patients had a GCS <8 - they were observed, with none requiring intubation, with no increased risk of adverse events including aspiration. Only one patient required intubation, after being admitted to the ward with a GCS of 12!

TAPNA 2013

Dear All,

On 2-3 May this year, Newcastle will host the TAPNA 2013 scientific meeting at the scenic Noah's on the beach hotel overlooking Newcastle beach.

TAPNA = Toxicology And Poisons Network Australasia

The Thursday and Friday will consist of plenary sessions and will feature state of the art talks and workshops on paracetamol poisoning by Marco Sivilotti, an Emergency physician and Clinical Toxicologist from Queens University Canada.

Professor Simon Brown from University of Western Australia will talk about bite and sting related anaphylaxis and other reactions.

Are reactions during snakebites caused by antivenom....OR venom....all will be revealed

Day 3 will be a separate satellite session

Registration out at the end of this month, more information including abstract submission on the website.

Seen an interesting or different tox case lately ? Submit an abstract. The best cases submitted will be selected for a short oral presentation with expert commentary provided by a Toxicologist - they get grilled not you !!

More information :

www.tapna.net (you might recognise the web server)

twitter 

facebook 

Google 

Cheers

Michael

 

How to enjoy summer

End of year RMO drinks Friday 18th January at Nobbys Lighthouse, $10 HRMOA members $30 not 

ED vs Ambos lawn bowls Thursday 7th February, Lowlands Bowling Club, 4-1130pm, $10, sausage sizzle, theme is "it was cool once"

Get into it people!

Severe pain protocol

Just came across a EMCRIT podcast on an ED pain management protocol that I thought was worth sharing...

Acute pain protocol:

  • Administer morphine 0.1 mg/kg IVP (If pt is > 55 y/o, substitute morphine 0.05 mg/kg IVP for this 1st dose)
    + diphenhydramine 0.5 mg/kg IVP
  • 7 minutes later the patient is asked, “Would you like more pain medicine?”
  • If the answer is yes, give a 2nd dose of morphine 0.05 mg/kg IVP
  • 7 minutes later, the patient is asked again, “Would you like more pain medicine?”
  • If the answer is yes, give a 3rd dose of morphine 0.05 mg/kg IVP
  • This continues every 7 minutes until the patient answers “no” to the question or the patient is asleep.

They use diphenhydramine as their anti-emetic but would be just as easy to subsitute metoclopramide or ondansetron.

The very interesting thing about this protocol is that the ED physician who came up with it states that in all the years its been running at his hospital he hasn't had to use naloxone.

Reference: EMCRIT Podcast 26: Patient controlled analgesia by Dr Edward Gentile

Thoughts?! 

Anticholinergics for abdominal pain?

In case any of you missed this:
Buscopan is not good for abdominal pain. Or at least, Paracetamol is better!
Prospective RCT
Participants - 132 patients in single ED setting
Intervention - PO Paracetamol
Comparison - vs. IV Buscopan + PO Paracetamol,  vs. IV Buscopan only
Outcomes - VAS scoring
Time - 30 and 60 mins
At 30 mins - no difference. At 60 mins, statistically significant reduction in pain was in the PO Paracetamol group only. In particular, sole treatment with Paracetamol seemed to better than Paracetamol + Buscopan!
Remington-Hobbs, Petts G, Harris T. Emergency department management of undifferentiated abdominal pain with hyoscine butylbromide and paracetamol: a randomised control trial.
Emergency Medicine Journal 2012; 29:989-994

ICC insertion in JHH

I posted a review a few weeks ago of an article I found in ANZ Journal of Surgery - here is another one! More critique of our Emergency department practice by our trauma colleagues. 
http://onlinelibrary.wiley.com/doi/10.1111/j.1445-2197.2012.06093.x/abstract
Introduction:  Intercostal catheters (ICC) are the standard management of chest trauma, but are associated with complications in up to 30%. The aim of this study was to evaluate errors in technique during ICC insertion to characterise the potential benefit of improved training programmes.
Methods:  Prospective audit of all ICC in trauma patients at a level 1 trauma centre for over 12 months. Exclusions were pigtail catheters and ICC inserted during thoracic surgery. Errors were identified from patient examination and chest imaging; they were defined as insertional, positional, incorrect size (<28 French) and lack of antibiotic prophylaxis. Ongoing complications unrelated to an error in technique, for example blocked tube, were not analysed.
Results:  Fifty-seven patients received a total of 94 ICC during the study period. Patients were predominantly male (77%), mean age of 40 ± 20 years, mean injury severity score 27 ± 13, mean abbreviated injury scale chest 3.8 ± 0.72. 86% were blunt trauma and 14% penetrating chest injuries. Thirty-six errors in technique occurred in 33 ICC insertions (38%). The most common errors were absence of prophylactic antibiotics (13%), ICC too far out (9%), kinked (6%) and wrong-sized ICC (5%). Emergency had a significantly greater frequency of errors than other specialties (67%, relative risk 2.11, P= 0.002). The majority of ICC were inserted by registrars, and registrars made a greater number of errors than fellows or consultants (relative risk 2.00, P= 0.02).
Discussion:  This study identified a large number of preventable errors for ICC insertion in trauma patients. Standardised institutional credentialing systems may be required to ensure adequate proficiency of trainees performing this procedure.
Insertional errors were defined as ICC too far out, kinked, inadequate fixation, insertion through previous ICC hole. 
Positional errors were defined as extrathoracic placement (obviously wrong) or in the wrong intercostal space. No mention of the safe triangle for ICC placement is made, which makes it hard to interpret if this is a useful statistic. 
Incorrect size was stated to be size less than 28 Fr (noting pigtails were excluded). 
Lack of antibiotic prophylaxis was the final error type. 
There are many assumptions that this study bases it's conclusions on, and, to be honest I think they invalidate most of the findings. The nature of the assessment of ICC insertion is too simplistic- a more detailed analysis could have been made from a prospective study. For example - the most common error noted - antibiotic prophylaxis for ICC insertion in trauma has not been well elucidated in the literature, however meta analyses seem to show benefit. Correct sterile technique has not been noted as an error although evidence shows it reduces the infection rate. 
Furthermore, evidence is becoming available to show that trauma dogma such as ICC size is becoming more untenable - particularly in stable small size traumatic pneumothorax which would normally score an ICC. I have not started doing it myself  but an informed decision to use a small size tube (or pigtail) may not necessarily be 'wrong'. According to the British Thoracic Society - 28Fr + is indicated for hemothorax only.
There a number of demographic issues in this study - 24% of ICC done in theatre - possibly intraoperatively, which is a very different prospect from a procedure in ED. 
My biggest problem with this study is there is no mention of actual complications- only 'errors' which were observed, with no clinical correlation (or relevance?). This may be me missing the point of the study, as the authors do state that they did not seek to note complications. I can't help but agree with their comment that direct observation would have been more likely to increase the pick up rate of error - breaches in sterile technique may have more relevance than antibiotic use! That said, the decreasing rate of error with increasing experience shows plausibility in the hypothesis. 
Emergency doctors of all levels of experience had more errors - I think we could all benefit from some standardised training in the emergency training curriculum.......... Before another specialty forces it upon us!

Fellowship teaching – CMH 20th Nov

 

The next fellowship teaching is at:

Calvary Mater, Tuesday 20th November, 9.30 - 11.30am, tutorial room.

 

Topics:

(i) 1st hour - VAQ / SAQ practice

(ii) 'Dancing with the stars' - Neuro exam practice with Ashanka (Neuro Fellow at CMH)

 

(Apologies Scott.  I know this belongs on 'Fellowship' page but couldn't get it to load up there..)